Muellner J and Schmidt KH (2023) Helicase activities of Rad5 and Rrm3 genetically interact in the prevention of recombinogenic DNA lesions in Saccharomyces cerevisiae. DNA Repair (Amst) 126:103488 PMID:37054652
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Doerfler L and Schmidt KH (2014) Exo1 phosphorylation status controls the hydroxyurea sensitivity of cells lacking the Pol32 subunit of DNA polymerases delta and zeta. DNA Repair (Amst) 24:26-36 PMID:25457771
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Mirzaei H and Schmidt KH (2012) Non-Bloom syndrome-associated partial and total loss-of-function variants of BLM helicase. Proc Natl Acad Sci U S A 109(47):19357-62 PMID:23129629
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Schmidt KH, et al. (2010) Defects in DNA lesion bypass lead to spontaneous chromosomal rearrangements and increased cell death. Eukaryot Cell 9(2):315-24 PMID:20008080
Vijayakumar S, et al. (2007) The C-terminal domain of yeast PCNA is required for physical and functional interactions with Cdc9 DNA ligase. Nucleic Acids Res 35(5):1624-37 PMID:17308348
Schmidt KH and Kolodner RD (2006) Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants. Proc Natl Acad Sci U S A 103(48):18196-201 PMID:17114288
Schmidt KH, et al. (2006) Control of translocations between highly diverged genes by Sgs1, the Saccharomyces cerevisiae homolog of the Bloom's syndrome protein. Mol Cell Biol 26(14):5406-20 PMID:16809776
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Schmidt KH and Kolodner RD (2004) Requirement of Rrm3 helicase for repair of spontaneous DNA lesions in cells lacking Srs2 or Sgs1 helicase. Mol Cell Biol 24(8):3213-26 PMID:15060145