N-terminal truncation of amino acids 1-186; PIP-box (PCNA binding site) and phosphorylation sites of the disordered tail deleted; DNA-damage response defective
List of references used specifically to curate the information in Overview section.
Syed S, et al. (2016) A Novel Rrm3 Function in Restricting DNA Replication via an Orc5-Binding Domain Is Genetically Separable from Rrm3 Function as an ATPase/Helicase in Facilitating Fork Progression. PLoS Genet. 12(12):e1006451