Mitochondrial markers of resistance to antibiotics erythromycin and neomycin were used to study mitochondrial recombination in Saccharomyces cerevisiae bifactorial crosses both in cis and trans configurations of markers. The following parameters of mitochondrial recombination have been examined: a) the asymmetrical transmission of parental classes, b) the frequency and the polarity of recombination and c) the effect of cell mating type on above listed parameters. The counting of results was carried out among random samples of zygotic diploid daughter colonies. The data obtained revealed the following features of mitochondrial recombination: 1. The origin of mitochondria controls all the parameters of mitochondrial recombination. 2. There are discovered two classes of mitochondrial crosses in relation to the polarity of recombination--a) a low polarity as "homosexual" (with types of mitochondria omega- X omega-) and b) a high polarity crosses as (heterosexual) (omega+ X omega+) 3. The linear arrangement of mitochondrial markers of the resistance to erythromycin and neomycin in relation to omega factor is suggested as omega-N-E. 4. The effect of cell mating type on the asymmetrical transmission of parental classes, frequency and polarity of recombination is established. 5. A new class of mitochondria is discovered that can transmit (or recombinate) with very high efficiency by ER-markers, but cannot transmit (or recombinate with) NR-markers.
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| Evidence ID | Analyze ID | Gene/Complex | Systematic Name/Complex Accession | Qualifier | Gene Ontology Term ID | Gene Ontology Term | Aspect | Annotation Extension | Evidence | Method | Source | Assigned On | Reference |
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| Evidence ID | Analyze ID | Gene | Gene Systematic Name | Phenotype | Experiment Type | Experiment Type Category | Mutant Information | Strain Background | Chemical | Details | Reference |
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| Evidence ID | Analyze ID | Gene | Gene Systematic Name | Disease Ontology Term | Disease Ontology Term ID | Qualifier | Evidence | Method | Source | Assigned On | Reference |
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| Evidence ID | Analyze ID | Regulator | Regulator Systematic Name | Target | Target Systematic Name | Direction | Regulation of | Happens During | Regulator Type | Direction | Regulation Of | Happens During | Method | Evidence | Strain Background | Reference |
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| Site | Modification | Modifier | Source | Reference |
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| Evidence ID | Analyze ID | Interactor | Interactor Systematic Name | Interactor | Interactor Systematic Name | Allele | Assay | Annotation | Action | Phenotype | SGA score | P-value | Source | Reference | Note |
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| Evidence ID | Analyze ID | Interactor | Interactor Systematic Name | Interactor | Interactor Systematic Name | Assay | Annotation | Action | Modification | Source | Reference | Note |
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| Complement ID | Locus ID | Gene | Species | Gene ID | Strain background | Direction | Details | Source | Reference |
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| Evidence ID | Analyze ID | Dataset | Description | Keywords | Number of Conditions | Reference |
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