Yeast Genetics and Molecular Biology 1998
College Park, Maryland
August 1998


Name: Yoshimoto, Hiroyuki
Mailing Address: Central Lab. for Key Tech., Kirin Brewery Co., Ltd., Kanazawa-ku, Yokohama-shi, 236-0004, Japan
Email Address: hyoshimoto@kirin.co.jp
Phone and Fax numbers: +81-45-788-7221, +81-45-788-4042

628

Mechanism of isoamyl alcohol and isoamyl acetate production in Saccharomyces cerevisiae .


Hiroyuki Yoshimoto (1) , Tomoyuki Fukushige (2), Toshihiko Yonezawa (2), Hidetaka Sone (1)
(1) Central Lab. for Key Tech., Kirin Brewery Co., Ltd., Kanazawa-ku, Yokohama-shi, 236-0004, Japan; (2) Gotemba Plant, Kirin-Seagram Ltd., Shibanuta, Gotemba, Shizuoka 412-0003 Japan

Isoamyl alcohol (i-AmOH) and isoamyl acetate (IsOAc) are important flavor components of yeast-fermented alcoholic beverages. i-AmOH is formed via the leucine synthetic pathway or via the Ehrlich mechanism from leucine by the branched-chain amino acid aminotransferase encoded by BAT1 and BAT2 genes. IsOAc is synthesized from i-AmOH and acetyl CoA by alcohol acetyltransferase encoded by the ATF1 gene. We show that the BAT2 gene product plays an important role for i-AmOH and IsOAc production by analyzing bat2 null mutants and transformants with multi-copy plasmids containing the BAT2 gene. As well, the effects of the nitrogen source on i-AmOH and IsOAc production are investigated by a fermentation test using synthetic medium which contains various concentrations of the nitrogen source. Results showed that the production of i-AmOH and IsOAc increased with increasing concentrations of the nitrogen source until the system was saturated. Further addition of the nitrogen source reduced the yields of i-AmOH and IsOAc. RNA blot analysis showed that transcription of BAT2 and LEU2 increased with increasing i-AmOH production and ATF1 transcription increased with increasing concentrations of the nitrogen source. These data suggest that transcription of BAT2 and LEU2 is co-regulated by the nitrogen source and ATF1 transcription is activated by the nitrogen source.


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