Yeast Genetics and Molecular Biology 1998
College Park, Maryland
August 1998


Name: Hon, Thomas
Mailing Address: Biochemistry, NYU Medical Center, 550 First Avenue, New York, NY 10016, USA
Email Address: hont01@popmail.med.nyu.edu
Phone and Fax numbers: 212-263-8159, 212-263-8166

053

Functional dissection of novel elements critical for heme regulation and transcriptional activity of HAP1.


Angela Hach, Thomas Hon , Li Zhang
Biochemistry, NYU Medical Center, 550 First Avenue , New York, NY 10016, USA

The yeast heme responsive transcriptional activator HAP1 serves as a paradigm for studying heme signaling in eukaryotic cells. HAP1 contains a DNA-binding domain, an activation domain and two heme regulatory domains. The HAP1 DNA-binding domain encompasses a C6 zinc cluster motif and a coiled-coil dimerization element typical of the yeast GAL4 family proteins. The heme regulatory domains--the heme domain and the HRM7 domain--contain respectively six and one heme responsive motifs (HRMs) that can bind directly to heme. We have carried out a systematic functional analysis of HAP1 elements required for heme regulation and transcriptional activation. Surprisingly, we found that the HRMs are not responsible for heme regulation of HAP1: HAP1 derivatives with the HRMs in the heme domain or all HRMs deleted are still repressed in the absence of heme and require heme for activation. Rather, three novel elements located at the end of the dimerization domain, in the beginning of the heme domain, and in the HRM7 domain are critical for heme regulation of HAP1. Further, we found that the coiled-coil dimerization element is dispensable for DNA binding and transcriptional activation at UAS1/CYC1, but is essential for transcriptional activation at UAS/CYC7. The HAP1 dimerization domain contains multiple elements that can substitute for each other in DNA binding and transcriptional activation. These unexpected findings provide novel insights into the mechanism governing heme activation of HAP1 and transcriptional activation by HAP1.


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