Yeast Genetics and Molecular Biology 1998
College Park, Maryland
August 1998


Name: Mills, Kevin
Mailing Address: Biology, MIT, 77 Massachusetts Ave, Cambridge, MA 02139, USA
Email Address: kmills@mit.edu
Phone and Fax numbers: (617) 253-6717, (617) 253-8699

038

The silent information regulator, Sir3p, relocalizes in response to a DNA damaging agent.


Kevin Mills , David Sinclair, Mitch McVey, Leonard Guarente
Biology, MIT, 77 Massachusetts Ave, Cambridge, MA 02139, USA

A form of position dependent gene repression in yeast, known as silencing, requires the function of the silent information regulators, Sir2p, Sir3p, and Sir4p. Position dependent repression is associated with the cryptic mating-type cassettes at HML and HMR , as well as telomeres and rDNA. Immunolocalization studies have shown Sir3p and Sir4p to reside predominantly at telomeres near the periphery of the nucleus. We have found that exposure of yeast cells to the DNA alkylating agent methyl methane sulfonate (MMS), results in a loss of telomeric silencing. Using indirect immunofluorescence we found that Sir3p redistributes in response to MMS. Sir3p requires entry into the S phase of the cell cycle for redistribution and undergoes a time dependent change in its staining pattern. Early in S phase Sir3p delocalizes from telomeres. Over time Sir3p reorganizes into a brightly staining nuclear spot and then into multiple (typically 10-50) spots throughout the nucleus that are not reminiscent of telomeric localization. The redistribution is not a consequence of cell cycle progression per se as cells synchronized but not treated with MMS do not show delocalization at any time during S phase. Continuation through S phase is not required for the intitial delocalization because cells arrested with the S phase inhibitor hydroxyurea still exhibit Sir3p delocalization near the G1/S phase transition. We postulate a role for Sir proteins in the repair of damaged DNA.


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