Yeast Genetics and Molecular Biology 1998
College Park, Maryland
August 1998


Name: Segev, Nava
Mailing Address: Pharm. Physiol. Sci., Univ. of Chicago, 947 East 58th St., Chicago, IL 60637, USA
Email Address: ns15@midway.uchicago.edu
Phone and Fax numbers: 773-702-3526, 773-702-3774

028

Genetic interactions between Ypt31/32 GTPases and Arf exchangeres.


Sara Jones (1), Gregory Jedd (1), Rick Kahn (2), Nava Segev (1)
(1) Pharm. Physiol. Sci., Univ. of Chicago, 947 East 58th St., Chicago, IL 60637, USA; (2) Dept. Bioch., Emory Univ.

Two families of GTPases, Arf and Ypt/rab, are key regulators of vesicular transport. While Arf proteins are implicated in vesicle budding from the donor compartment, Ypt/rab proteins are involved in the targeting of vesicles to the acceptor compartment. Recently, we have shown a role for Ypt31/32p in exit from the yeast trans-Golgi, suggesting a function for Ypt/rab proteins in vesicle budding as well. Here we report the identification of a new member of the Sec7-domain family, SYT1, as a high copy suppressor of a ypt31/32 mutation. Several proteins that belong to the Sec7-domain family, including the yeast Gea1p, have been shown recently to stimulate nucleotide exchange by Arf GTPases. Sec7p itself has an important role in the yeast secretory pathway. However, its mechanism of action is not yet understood. We show that SEC7 also exhibit reciprocal genetic interactions with the YPT31/32 genes. Biochemical assays demonstrate the ability of Sec7p and Syt1p to promote nucleotide exchange on Arf proteins, but not on Ypt GTPases. The Sec7-domain of Sec7p is sufficient for this activity. These results demonstrate that the ability to act as Arf exchangers is a general property of all Sec7-domain proteins in yeast. The genetic interactions observed between Arf exchangers and Ypt31/32p suggest the existence of a Ypt-Arf GTPase cascade in the secretory pathway. In addition, the genetic interaction of Arf exchangers with Ypt31/32p supports the idea of a role for Ypt GTPases in vesicle budding.


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