Yeast Genetics and Molecular Biology 1998
College Park, Maryland
August 1998


Name: Grewal, SHIV I. S.
Mailing Address: Developmental Genetics Section, ABL- Basic Research Program, P.O. Box B, Frederick, MD 21702-1201, USA
Email Address: grewals@ncifcrf.gov
Phone and Fax numbers: 301-846-5150, 301-846-6911

012

Histone Deacetylase Homologs Regulate Epigenetic Inheritance of Transcriptional Silencing and Chromosome Segregation in Fission Yeast.


Shiv Grewal , Mike Bonaduce, Amar Klar
Developmental Genetics Section, ABL- Basic Research Program, P.O. Box B, Frederick, MD 21702-1201, USA

Position-effect control at the silent mat2-mat3 interval and at centromeres and telomeres in fission yeast is suggested to be mediated through assembly of heterochromatin-like structures. Therefore, trans-acting genes that affect silencing may encode either chromatin proteins, factors that modify them or factors that affect chromatin assembly. Here, we report the identification of an essential gene, clr6 (cryptic loci regulator), encoding a putative histone deacetylase, which when mutated affects epigenetically-maintained repression at the mat2-mat3 region and at centromeres and reduces the fidelity of chromosome segregation. Furthermore, we show that the Clr3 protein, which regulates silencing at the mat2-mat3 region and at centromeres and telomeres, also shares strong homology to known histone deacetylases. Genetic analyses indicate that histone deacetylation in fission yeast might be regulated by at least two overlapping mechanisms. We also found that transient inhibition of histone deacetylase activity by trichostatin A results in the increased missegregation of chromosomes in subsequent generations and, remarkably, alters the imprint at the mat locus, causing the heritable conversion of the repressed epigenetic state to the expressed state. This work supports the model that the level of histone deacetylation has a role in the assembly of repressive heterochromatin and provide insight into the mechanism of epigenetic inheritance. Research sponsored by National Cancer Institute, DHHS, under contract with ABL


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