Yeast Genetics and Molecular Biology 1998
College Park, Maryland
August 1998


Name: Homesley, Lisa
Mailing Address: Biochemistry, Cornell University, 327 Biotech Bldg., Ithaca, NY 14853, United States
Email Address: LH33@cornell.edu
Phone and Fax numbers: (607) 255-2447, (607) 255-2428

002

Mutual suppression between two initiation mutants.


Lisa Homesley , Bik-Kwoon Tye
Biochemistry, Cornell University, 327 Biotech Bldg., Ithaca, NY 14853, United States

Initiation of DNA replication in S. cerevisiae involves a homologous family of proteins called the m ini c hromosome m aintainance (Mcm) proteins. There are six of these proteins (Mcm2-7) and although their function in initiation is not known, they have been shown to act as a complex and interact with such proteins known to act at origins of DNA replication as Orc and Cdc7/Dbf4. They are bound to chromatin in a cell cycle dependent manner. Another Mcm protein, Mcm 10, was recently characterized in our lab. Although identified in the same screen in which some of the other Mcm proteins (Mcm2,3,5) were also identified, Mcm10 is not a member of the homologous family. Its role in initiation is also not well understood, but the protein binds single stranded DNA and may be involved in several aspects of initiation and replication. I will discuss an interesting genetic interaction between the mutants mcm10-1 and cdc47-1 (an allele of MCM7 ) which are both temperature sensitive. The double mutant is viable at the restrictive temperature for both mutations, showing a kind of mutual suppression . This mutual suppression is also seen between mcm10-1 and a mutant allele of MCM 5 . I will present a chracterization of the double mutant by 2D DNA gel, FACS analysis, and MCM assay. I will also relate results of a two-hybrid study and gene deletion to test possible hypotheses for the mechanism of mutual suppression, and present a model to explain these fascinating results. Understanding the cause of this mutual suppresion should shed some light on the function of these Mcm proteins in DNA replication initiation.


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