Yeast Genetics and Molecular Biology 2002
University of Wisconsin
Madison, Wisconsin USA
July 30 - August 4, 2002


Name: Hasek, Jiri
Mailing Address: Cell and Mol. Microbiology, Inst.Microbiology ASCR, Videnska 1083, Prague 4, 14220, Czech Republic
Email Address: hasek@biomed.cas.cz
Phone & FAX numbers: 420241062503 & 420241062501

Abstract #99


Session Title: Cell Biology: Cytoskeleton
Presentation: Poster
Topic: Cell Biology

Rpg1/Tif32p accumulates at sites of the localized growth.
Jiri Hasek, Katerina Malinska, Ivana Janatova
Cell and Mol. Microbiology, Inst.Microbiology ASCR, Videnska 1083, Prague 4, 14220, Czech Republic

Rpg1/Tif32p is the essential subunit of the translation initiation factor 3 core complex (eIF3) of Saccharomyces cerevisiae. We have shown that Rpg1p is partially localized to microtubules and physically interacts with the actin-associated protein Sla2/End4/Mop2. In this work, we co-localized Rpg1p with Sla2p, Hcr1p and the endoplasmic reticulum marker Elo3-GFP in wild type cells and sla2 deletion mutants using the immunofluorescence microscopy. Proteins Hcr1, Sla2 and Rpg1 were obviously co-localized at the tip of the emerging bud. In comparison to wild type cells, the localization of Rpg1p to microtubules seemed to be prominent in the sla2 deletion mutant. To monitor distribution of Rpg1p in living cells, we prepared various C-terminal Rpg1-GFP fusions. In general, living wild type cells expressing Rpg1-GFP from the RPG1 promoter displayed almost homogeneous fluorescence in the cytoplasm. Nevertheless, in some living cells we clearly observed filamentous structures associated with patches of Rpg1-GFP near the bud tip and the bud neck. These patches were dominant in cells overproducing Rpg1-GFP. The accumulation of Rpg1-GFP near the tip of the emerging bud was prominent in living cells of the cdc28-4 ts mutant shifted back from 37°C to 25°C. We suggest that the accumulation of Rpg1p at certain sites at the cell periphery is associated with the establishment of the localized growth. This work was financed by grant GACR 204/02/1424 to JH.


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