Yeast Genetics and Molecular Biology 2002
University of Wisconsin
Madison, Wisconsin USA
July 30 - August 4, 2002


Name: Dunham, Maitreya J.
Mailing Address: Department of Genetics, Stanford Univ. Medical School, 300 Pasteur Dr., Stanford, CA 94305-5120, USA
Email Address: maitreya@stanford.edu
Phone & FAX numbers: 650-723-4973

Abstract #31


Session Title: Comparative Genetics and Evolution
Session Time: Thursday, August 1 -- 11:00AM - 12:30PM
Presentation: Platform
Topic: Global Analysis

Characteristic genome rearrangements accompany experimental evolution.
Maitreya J. Dunham (1), Hassan Badrane (2), Patrick O. Brown (3), R. Frank Rosenzweig (4), David Botstein (1)
(1) Department of Genetics, Stanford Univ. Medical School, 300 Pasteur Dr., Stanford, CA 94305-5120, USA; (2) University of Florida College of Medicine; (3) HHMI, Biochemistry Department, Stanford University Medical School; (4) University of Montana

Paquin, Adams, and Rosenzweig followed the evolution of eight independent cultures of S. cerevisiae diploids for hundreds of generations in glucose-limited chemostats. Ferea et al. (1999) reported changes in global gene expression in three of these strains consistent with a shift from reliance on fermentation to respiration. Here we describe chromosomal rearrangements that occurred during the evolution of six of the eight cultures, by the technique of Comparative Genomic Hybridization on DNA microarrays. As with the gene expression results, the genome rearrangements are surprisingly consistent. Three evolved strains contain amplifications of regions of chromosome 4 that include multiple high affinity hexose transporters; one of these is limited to HXT6 and HXT7, as described previously by Brown et al. (1998). Three strains contain nested deletions on chromosome 15. Finally, and most strikingly, three strains have a deletion on chromosome 14 that begins near CIT1, which encodes the first enzyme in the tricarboxylic acid cycle. Other amplification and deletion events were also observed. Most, if not all, of these rearrangements occur in the immediate vicinity of sequences related to transposons. Two of the breakpoints near CIT1 have been cloned and found to be fused to a complete transposon, even though the wildtype sequence is only a transposon end. The similarity of these events to those regularly observed in other systems, especially cancer progression, will be noted.


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