Yeast Genetics and Molecular Biology 2002
University of Wisconsin
Madison, Wisconsin USA
July 30 - August 4, 2002


Name: Kuchler, Karl
Mailing Address: Institute Medical Biochemistry, Vienna BioCenter, Dr. Bohr-Gasse 9/2, Vienna, A-1030, Austria
Email Address: kaku@mol.univie.ac.at
Phone & FAX numbers: +43-1-4277-61807 & +43-1-4277-9618
URL: http://www.at.embnet.org/molg/kklab.htm

Abstract #20


Session Title: Global Analysis of Gene Expression
Session Time: Wednesday, July 31 -- 2:00PM - 3:30PM
Presentation: Platform
Topic: Cell Biology

A novel transcription factor, War1p, mediates adaptation to weak acid stress by controlling expression of the yeast Pdr12p ATP-binding cassette (ABC) transporter.
Yasmine Mamnun (1), Angelika Kren (1), Bettina Bauer (1), Christoph Schüller (1), Chantal Schwartz (1), Mehdi Mollapour (2), Peter Piper (2), Karl Kuchler (3)
(1) Institute Medical Biochemistry, Vienna BioCenter, Dr. Bohr-Gasse 9/2, Vienna, A-1030, Austria; (2) University College London, Department of Biochemistry & Molecular Biology, Gower Street, London WC1E 6BT, United Kingdom; (3) Institute of Medical Biochemistry, University & BioCenter of Vienna, Dr. Bohr-Gasse 9/2, A-1030 Vienna, Austria, Phone: +43-1-4277-61807, FAX: +43-1-4277-9618, e-mail: kaku@mol.univie.ac.at

The S. cerevisiae genome harbors 30 genes encoding ATP-binding cassette (ABC) proteins. We demonstrated that the Pdr12p ABC pump mediates resistance to carboxylate anions such as sorbate and benzoate, and that PDR12 is tightly regulated by adverse conditions such weak acid stress. However, specific regulators have not been identified. We have thus employed a genetic screen to isolate mutants displaying increased weak acid sensitivity. In addition, we screened the deletion collection to identify genes mediating weak acid tolerance. Finally, transcriptome analysis using DNA microarrays established a subset of genes involved in weak acid stress adaptation. Taken together, our work uncovered a novel transcriptional regulator, War1p, which is required for the induction of PDR12 after stress exposure. War1p constitutively localizes to the nucleus, and becomes rapidly phosphorylated and perhaps activated upon stress challenge. The PDR12 induction is controlled through a novel cis-acting element, the weak acid response element (WARE). In vivofootprinting experiments and promoter deletions identified and demonstrated the existence of three distinct WAREs in the PDR12 promoter, at least two of which are necessary and sufficient for PDR12 expression regulation. The War1p transcription factor controls a rather small but distinct weak acid response regulon dedicated to weak organic acid stress adaptation. War1p represents the first stress-specific regulator for a eukaryotic ABC gene.


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