Yeast Genetics and Molecular Biology 2002
University of Wisconsin
Madison, Wisconsin USA
July 30 - August 4, 2002


Name: Verwaal, René
Mailing Address: Molecular Cell Biology, Utrecht University, Padualaan 8, Utrecht, 3584 CH, The Netherlands
Email Address: r.verwaal@bio.uu.nl
Phone & FAX numbers: +31302532598 & +31302513655

Abstract #129


Session Title: Cell Biology: Growth and Metabolism
Presentation: Poster
Topic: Cell Biology

Expression of HXT5 is determined by growth rates in Saccharomyces cerevisiae .
René Verwaal, Hans Paalman, Rick Kapur, Astrid Hogenkamp, Arie Verkleij, Theo Verrips, Johannes Boonstra
Molecular Cell Biology, Utrecht University, Padualaan 8, Utrecht, 3584 CH, The Netherlands

In Saccharomyces cerevisiae, hexose transporters mediate transport of glucose across the plasma membrane. A multigene family that includes HXT1-17, GAL2, SNF3 and RGT2 encodes these transporters. The proteins encoded by HXT1-4 and HXT6-7, whose expression is regulated by extracellular glucose concentrations, are the main contributors to glucose transport. One of the remaining hexose transporters, encoded by HXT5, also has glucose transport capacity. However, we found that expression of HXT5 mRNA and Hxt5 protein was not regulated by the extracellular glucose concentration, but by the growth rate of cells. During batch growth, expression of HXT5 was induced prior to glucose depletion, simultaneously with a decrease in the growth rate of cells. Increasing the temperature or osmolarity, conditions that are known to decrease growth, induced expression of HXT5. Decreasing the growth rate to low rates in a fed-batch culture also resulted in expression of HXT5. Definite proof that showed that indeed the growth rate determined expression of HXT5 was obtained from N-limited continuous culture experiments, where HXT5 was expressed only at low growth rates (D<0.1). The promoter of HXT5 contains putative regulatory elements including one PDS element, two STRE elements and two HAP2/3/4/5 binding sites, which may be involved in the regulation of HXT5 expression. Therefore, we are currently determining whether these putative elements contribute to expression of HXT5 during low growth rates.


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