Yeast Genetics and Molecular Biology 2000
University of Washington
Seattle, Washington USA
July 2000


Name: Catlett, Natalie
Mailing Address: Biochemistry, University of Iowa, 51 Newton Road, Iowa City, IA 52242, USA
Email Address: natalie-catlett@uiowa.edu
Phone & FAX numbers: 1(319)335-8548 & 1(319)335-9570

#042

Identifiacation of a putative vacuole-binding region in the Myo2p tail domain.
Natalie Catlett, Kuniko Ishikawa, Jason Duex, Lois Weisman
Biochemistry, University of Iowa, 51 Newton Road, Iowa City, IA 52242, USA

The Saccharomyces cerevisiae myosin-V, Myo2p, is essential for polarized growth, probably through transport of secretory vesicles to the developing bud. Myo2p is also required for movement of a portion of the mother cell vacuole to the bud, a process not essential for growth. The carboxyl-terminal globular tail of class V myosins is proposed to interact with receptors on cargo organelles. Through random mutagenesis of this domain, we obtained seven unique single point mutants with defects in vacuole inheritance, but not polarized growth. One of these mutants was myo2-2 (G1248D), the others clustered to D1297, L1301, N1304, and N1307, amino acids located on one face of a predicted alpha-helix. Conversely, we identified a second region of the Myo2p tail required only for polarized growth. A deletion of amino acids 1459-1491, cannot support growth as the sole myo2 gene, but can support vacuole inheritance in myo2-2 cells. This observation that specific regions of the globular tail are required for different functions of Myo2p predicts the presence of cargo-specific receptors for Myo2p. In order to identify the vacuolar receptor for Myo2p, we screened for both multicopy and genomic suppressors of the myo2 vacuole inheritance mutants identified above. We have found two open reading frames that partially suppress the myo2-2 vacuole inheritance defect when overexpressed. In addition, we have identified one dominant suppressor mutant.


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