Yeast Genetics and Molecular Biology 2000
University of Washington
Seattle, Washington USA
July 2000


Name: Guttmann-Raviv, Noga
Mailing Address: Biology, Technion, Technion City, Haifa, 32000, Israel
Email Address: ykassir@tx.technion.ac.il
Phone & FAX numbers: 972-4-829-4323 & 972-4-822-5153

#031

Ime2, a meiosis-specific kinase in budding yeast, is required for destabilization of its transcriptional activator, Ime1.
Noga Guttmann-Raviv, Yona Kassir
Biology, Technion, Technion City, Haifa, 32000, Israel

In the budding yeast Saccharomyces cerevisiae entry and successful completion of meiosis depends on two positive regulators Ime1 and Ime2. Ime1 is a transcriptional activator that is required for the transcription of IME2, a serine threonine protein kinase. We show that Ime1 is not detected in vegetative cultures with either glucose or acetate as the sole carbon source. A transient induction in the steady state level of Ime1 is observed in meiotic cultures. This transient expression correlates with the pattern of IME1 mRNA. Pulse chase and Western analysis reveal that Ime1 is a non-stable protein. This stability depends on the kinase activity of Ime2. Haploid cells overexpressing IME2 show a decrease in the level of Ime1, whereas diploid cells deleted for IME2 show an increase in the level of Ime1, even in vegetative media. Using a temperature sensitive mutation in one of the ATPase genes of the proteosome, RPT2, we demonstrate that Ime1 is degraded by the 26S proteosome. We also show that Ime2 itself is an extremely non-stable protein, whose expression in vegetative cultures is toxic. Thus a negative feedback loop ensures that the activity of Ime1 will be restricted to a short window span. This loop is essential for efficient meiosis.


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