Yeast Genetics and Molecular Biology 2000
University of Washington
Seattle, Washington USA
July 2000


Name: Philpott, Caroline C.
Mailing Address: Liver Diseases Section, NIDDK, NIH, Bldg.10, Rm9B16, Bethesda, MD 20892-1800, USA
Email Address: carolinep@intra.niddk.nih.gov
Phone & FAX numbers: (301) 435-4018 & (301) 402-0491

#010

Siderophore-mediated iron uptake in Saccharomyces cerevisiae .
Cheol-Won Yun (1), Tracy Ferea (2), Orly Ardon (3), Patrick O. Brown (2), David Botstein (2), Robert E. Moore (4), Jerry Kaplan (3), Caroline C. Philpott (1)
(1) Liver Diseases Section, NIDDK, NIH, Bldg.10, Rm 9B16, Bethesda, MD 20892-1800, USA; (2) Stanford University School of Medicine, Stanford, CA ; (3) University of Utah, Salt Lake City, UT; (4) University of Wisconsin, Oshkosh, WI

In budding yeast, uptake of iron is largely controlled by the transcription factor Aft1. cDNA microarrays were used to identify new iron- and AFT1-regulated genes. Four genes regulated as part of the AFT1-regulon (ARN1-4) encoded members of a subfamily of the major facilitator superfamily of transporters. These proteins have 14 predicted transmembrane domains and were from 26% - 53% identical at the amino acid level. ARN1-4 were found to facilitate the transport of siderophores: low molecular weight compounds that specifically bind ferric iron. Our data indicate that S. cerevisiae maintains two systems of siderophore-mediated iron uptake, one occurring at the plasma membrane and the other occurring in an intracellular compartment. The plasma membrane-based system required the reduction of the siderophore-bound iron by the surface reductases FRE1, FRE2, and FRE3. Uptake of reduced siderophore-iron required the high-affinity, ferrous iron transport complex encoded by FET3 and FTR1. A second system of uptake required the Arn transporters. We determined the specificity of different siderophores for individual Arn transporters. Arn1p and Arn3p were localized to intracellular vesicles that co-sediment with the late endosomal protein Pep12p. Siderophore uptake was deficient in an endocytosis mutant, end4. The intracellular distribution of Arn1p did not change in the end4 mutant, however, suggesting that siderophores are delivered to the Arn transporters through an endocytic pathway.
cerevisiae can be exploited to clone nucleoside transporters from other species or from tissues of pharmacological relevance.


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