APN1/YKL114C Literature Guide Help

Other names published for APN1: DNA-(apurinic or apyrimidinic site) lyase APN1, YKL114C

APN1 - Strains/Constructs (107)

ReferenceOther Genes Addressed
Ben-Shitrit T, et al.  (2012) Systematic identification of gene annotation errors in the widely used yeast mutation collections.LID - 10.1038/nmeth.1890 [doi] Nat Methods ()
Collura A, et al.  (2012) Abasic sites linked to dUTP incorporation in DNA are a major cause of spontaneous mutations in absence of base excision repair and Rad17-Mec3-Ddc1 (9-1-1) DNA damage checkpoint clamp in Saccharomyces cerevisiae. DNA Repair (Amst) 11(3):294-303
Lazzaro F, et al.  (2012) RNase H and postreplication repair protect cells from ribonucleotides incorporated in DNA. Mol Cell 45(1):99-110
Morris LP, et al.  (2012) Saccharomyces cerevisiae Apn1 mutation affecting stable protein expression mimics catalytic activity impairment: implications for assessing DNA repair capacity in humans. DNA Repair (Amst) 11(9):753-65
Reis AM, et al.  (2012) Targeted detection of in vivo endogenous DNA base damage reveals preferential base excision repair in the transcribed strand. Nucleic Acids Res 40(1):206-19
Sobol Z and Schiestl RH  (2012) Intracellular and extracellular factors influencing Cr(VI) and Cr(III) genotoxicity. Environ Mol Mutagen 53(2):94-100
Wurtele H, et al.  (2012) Histone H3 lysine 56 acetylation and the response to DNA replication fork damage. Mol Cell Biol 32(1):154-72
Yibmantasiri P, et al.  (2012) Molecular basis for fungicidal action of neothyonidioside, a triterpene glycoside from the sea cucumber, Australostichopus mollis. Mol Biosyst 8(3):902-12
Yu S, et al.  (2012) Compromised cellular responses to DNA damage accelerate chronological aging by incurring cell wall fragility in Saccharomyces cerevisiae. Mol Biol Rep 39(4):3573-83
Kim N, et al.  (2011) The dCMP transferase activity of yeast Rev1 is biologically relevant during the bypass of endogenously generated AP sites. DNA Repair (Amst) 10(12):1262-71
Lada AG, et al.  (2011) Mutator effects and mutation signatures of editing deaminases produced in bacteria and yeast. Biochemistry (Mosc) 76(1):131-46
Ochi Y, et al.  (2011) Sensitive detection of chemical-induced genotoxicity by the Cypridina secretory luciferase reporter assay, using DNA repair-deficient strains of Saccharomyces cerevisiae. Yeast 28(4):265-78
Redrejo-Rodriguez M, et al.  (2011) New Insights in the Removal of the Hydantoins, Oxidation Product of Pyrimidines, via the Base Excision and Nucleotide Incision Repair Pathways. PLoS One 6(7):e21039
Svensson JP, et al.  (2011) Genomic phenotyping of the essential and non-essential yeast genome detects novel pathways for alkylation resistance. BMC Syst Biol 5(1):157
Yang J, et al.  (2011) Cell size and growth rate are major determinants of replicative lifespan. Cell Cycle 10(1)
Yang J, et al.  (2011) Cell size and growth rate are major determinants of replicative lifespan. Cell Cycle 10(1):144-55
Daley JM, et al.  (2010) Genetic interactions between HNT3/Aprataxin and RAD27/FEN1 suggest parallel pathways for 5' end processing during base excision repair. DNA Repair (Amst) 9(6):690-9
Degrandi TH, et al.  (2010) Evaluation of the cytotoxicity, genotoxicity and mutagenicity of diphenyl ditelluride in several biological models. Mutagenesis 25(3):257-69
Kim N and Jinks-Robertson S  (2010) Abasic sites in the transcribed strand of yeast DNA are removed by transcription-coupled nucleotide excision repair. Mol Cell Biol 30(13):3206-15
Manikova D, et al.  (2010) Investigations on the role of base excision repair and non-homologous end-joining pathways in sodium selenite-induced toxicity and mutagenicity in Saccharomyces cerevisiae. Mutagenesis 25(2):155-62
Matuo R, et al.  (2010) DNA repair pathways involved in repair of lesions induced by 5-fluorouracil and its active metabolite FdUMP. Biochem Pharmacol 79(2):147-53
Northam MR, et al.  (2010) Participation of DNA polymerase {zeta} in replication of undamaged DNA in Saccharomyces cerevisiae. Genetics 184(1):27-42
Ochi Y, et al.  (2010) Sensitive detection of chemical-induced genotoxicity by the Cypridina secretory luciferase reporter assay, using DNA repair-deficient strains of Saccharomyces cerevisiae. Yeast ()
Steinboeck F, et al.  (2010) The relevance of oxidative stress and cytotoxic DNA lesions for spontaneous mutagenesis in non-replicating yeast cells. Mutat Res 688(1-2):47-52
Swartzlander DB, et al.  (2010) Regulation of base excision repair: Ntg1 nuclear and mitochondrial dynamic localization in response to genotoxic stress. Nucleic Acids Res 38(12):3963-74
Acevedo-Torres K, et al.  (2009) Requirement of the Saccharomyces cerevisiae APN1 gene for the repair of mitochondrial DNA alkylation damage. Environ Mol Mutagen 50(4):317-27
Kaniak A, et al.  (2009) Msh1p counteracts oxidative lesion-induced instability of mtDNA and stimulates mitochondrial recombination in Saccharomyces cerevisiae. DNA Repair (Amst) 8(3):318-29
Kim N and Jinks-Robertson S  (2009) dUTP incorporation into genomic DNA is linked to transcription in yeast. Nature 459(7250):1150-3
Pogorzala L, et al.  (2009) Evidence that msh1p plays multiple roles in mitochondrial base excision repair. Genetics 182(3):699-709
van der Kemp PA, et al.  (2009) PCNA monoubiquitylation and DNA polymerase eta ubiquitin-binding domain are required to prevent 8-oxoguanine-induced mutagenesis in Saccharomyces cerevisiae. Nucleic Acids Res 37(8):2549-59