Other names published for RAD53: LSD1, MEC2, SPK1, YPL153C
RAD53 LITERATURE TOPICS
- Curated Literature
- Genetics/Cell Biology
- Nucleic Acid Information
- Gene Product Information
- Protein Physical Properties
- Protein Processing/Modification/Regulation
- Protein Sequence Features
- Protein-Nucleic Acid Interactions
- Protein-protein Interactions
- Protein/Nucleic Acid Structure
- Substrates/Ligands/Cofactors
- Related Genes/Proteins
- Research Aids
- Genome-wide Analysis
- Proteome-wide Analysis
- Other Topics
- Additional Information
RAD53 - Protein Processing/Modification/Regulation (203)
| Reference | Other Genes Addressed |
|---|---|
| Berens TJ and Toczyski DP (2012) Colocalization of Mec1 and Mrc1 is sufficient for Rad53 phosphorylation in vivo. Mol Biol Cell 23(6):1058-67 | |
| Castellano-Pozo M, et al. (2012) R-loops cause replication impairment and genome instability during meiosis. EMBO Rep 13(10):923-9 | |
| Davidson MB, et al. (2012) Endogenous DNA replication stress results in expansion of dNTP pools and a mutator phenotype. EMBO J 31(4):895-907 | |
| Fukunaga K, et al. (2012) Subtelomere-binding protein Tbf1 and telomere-binding protein Rap1 collaborate to inhibit localization of the Mre11 complex to DNA ends in budding yeast. Mol Biol Cell 23(2):347-59 | |
| Lazzaro F, et al. (2012) RNase H and postreplication repair protect cells from ribonucleotides incorporated in DNA. Mol Cell 45(1):99-110 | |
| Srividya I, et al. (2012) Yeast transcription termination factor rtt103 functions in DNA damage response. PLoS One 7(2):e31288 | |
| Tkach JM, et al. (2012) Dissecting DNA damage response pathways by analysing protein localization and abundance changes during DNA replication stress. Nat Cell Biol 14(9):966-76 | |
| Wang G, et al. (2012) Multiple phosphorylation of Rad9 by CDK is required for DNA damage checkpoint activation. Cell Cycle 11(20) | |
| Wang G, et al. (2012) Multiple phosphorylation of Rad9 by CDK is required for DNA damage checkpoint activation. Cell Cycle 11(20):3792-800 | |
| Ward TA, et al. (2012) Components of a fanconi-like pathway control pso2-independent DNA interstrand crosslink repair in yeast. PLoS Genet 8(8):e1002884 | |
| Wurtele H, et al. (2012) Histone H3 lysine 56 acetylation and the response to DNA replication fork damage. Mol Cell Biol 32(1):154-72 | |
| van Deursen F, et al. (2012) Mcm10 associates with the loaded DNA helicase at replication origins and defines a novel step in its activation. EMBO J 31(9):2195-206 | |
| Clemente-Ruiz M, et al. (2011) Histone H3K56 acetylation, CAF1, and Rtt106 coordinate nucleosome assembly and stability of advancing replication forks. PLoS Genet 7(11):e1002376 | |
| Duch A, et al. (2011) A Dbf4 Mutant Contributes to Bypassing the Rad53-mediated Block of Origins of Replication in Response to Genotoxic Stress. J Biol Chem 286(4):2486-91 | |
| Dyavaiah M, et al. (2011) Autophagy-Dependent Regulation of the DNA Damage Response Protein Ribonucleotide Reductase 1. Mol Cancer Res 9(4):462-475 | |
| Engels K, et al. (2011) 14-3-3 proteins regulate exonuclease 1-dependent processing of stalled replication forks. PLoS Genet 7(4):e1001367 | |
| Kubota T, et al. (2011) Quantitative proteomic analysis of chromatin reveals that Ctf18 acts in the DNA replication checkpoint. Mol Cell Proteomics 10(7):M110.005561 | |
| Lee MT, et al. (2011) The SUMO Isopeptidase Ulp2p Is Required to Prevent Recombination-Induced Chromosome Segregation Lethality following DNA Replication Stress. PLoS Genet 7(3):e1001355 | |
| Leon Ortiz AM, et al. (2011) Srs2 overexpression reveals a helicase-independent role at replication forks that requires diverse cell functions. DNA Repair (Amst) 10(5):506-17 | |
| Minard LV, et al. (2011) SWI/SNF and Asf1 Independently Promote Derepression of the DNA Damage Response Genes under Conditions of Replication Stress. PLoS One 6(6):e21633 | |
| Minard LV, et al. (2011) Transcriptional Regulation by Asf1: NEW MECHANISTIC INSIGHTS FROM STUDIES OF THE DNA DAMAGE RESPONSE TO REPLICATION STRESS. J Biol Chem 286(9):7082-92 | |
| Navadgi-Patil VM, et al. (2011) The unstructured C-terminal tail of yeast Dpb11 (human TopBP1) protein is dispensable for DNA replication and the S phase checkpoint but required for the G2/M checkpoint. J Biol Chem 286(47):40999-1007 | |
| Puddu F, et al. (2011) Sensing of Replication Stress and Mec1 Activation Act through Two Independent Pathways Involving the 9-1-1 Complex and DNA Polymerase epsilon. PLoS Genet 7(3):e1002022 | |
| Rai R, et al. (2011) Small Ubiquitin-related Modifier Ligase Activity of Mms21 Is Required for Maintenance of Chromosome Integrity during the Unperturbed Mitotic Cell Division Cycle in Saccharomyces cerevisiae. J Biol Chem 286(16):14516-30 | |
| Reha-Krantz LJ, et al. (2011) Drug-sensitive DNA polymerase d reveals a role for mismatch repair in checkpoint activation in yeast. Genetics 189(4):1211-24 | |
| Robert T, et al. (2011) HDACs link the DNA damage response, processing of double-strand breaks and autophagy. Nature 471(7336):74-9 | |
| Smith JS, et al. (2011) Rudimentary G-quadruplex-based telomere capping in Saccharomyces cerevisiae. Nat Struct Mol Biol 18(4):478-85 | |
| Sundararajan R and Freudenreich CH (2011) Expanded CAG/CTG Repeat DNA Induces a Checkpoint Response That Impacts Cell Proliferation in Saccharomyces cerevisiae. PLoS Genet 7(3):e1001339 | |
| Yeung M and Durocher D (2011) Srs2 enables checkpoint recovery by promoting disassembly of DNA damage foci from chromatin. DNA Repair (Amst) 10(12):1213-22 | |
| Bazzi M, et al. (2010) Dephosphorylation of {gamma}H2A by Glc7/Protein Phosphatase 1 Promotes Recovery from Inhibition of DNA Replication. Mol Cell Biol 30(1):131-45 |




