| Montanari A, et al. (2010) Aminoacyl-tRNA synthetases are multivalent suppressors of defects due to human equivalent mutations in yeast mt tRNA genes. Biochim Biophys Acta 1803(9):1050-1057
|
|
| Wang X, et al. (2010) Combination of the loss of cmnm5U34 with the lack of s2U34 modifications of tRNALys, tRNAGlu, and tRNAGln altered mitochondrial biogenesis and respiration. J Mol Biol 395(5):1038-48
|
|
| Chan PP and Lowe TM (2009) GtRNAdb: a database of transfer RNA genes detected in genomic sequence. Nucleic Acids Res 37(Database issue):D93-7
|
|
| De Luca C, et al. (2009) Can yeast be used to study mitochondrial diseases? Biolistic tRNA mutants for the analysis of mechanisms and suppressors. Mitochondrion 9(6):408-17
|
|
| Montanari A, et al. (2008) Yeast as a model of human mitochondrial tRNA base substitutions: Investigation of the molecular basis of respiratory defects. RNA 14(2):275-83
|
|
| De Luca C, et al. (2006) Mutations in yeast mt tRNAs: specific and general suppression by nuclear encoded tRNA interactors. Gene 377():169-76
|
|
| Jones CI, et al. (2006) A counterintuitive Mg2+-dependent and modification-assisted functional folding of mitochondrial tRNAs. J Mol Biol 362(4):771-86
|
|
| Feuermann M, et al. (2003) The yeast counterparts of human 'MELAS' mutations cause mitochondrial dysfunction that can be rescued by overexpression of the mitochondrial translation factor EF-Tu. EMBO Rep 4(1):53-8
|
|
| Foury F, et al. (1998) The complete sequence of the mitochondrial genome of Saccharomyces cerevisiae. FEBS Lett 440(3):325-31
|
|