TPD3/YAL016W Literature Guide Help

Other names published for TPD3: FUN32, YAL016W

TPD3 - Primary Literature (23)

ReferenceOther Genes Addressed
de Clare M and Oliver SG  (2013) Copy-number variation of cancer-gene orthologs is sufficient to induce cancer-like symptoms in Saccharomyces cerevisiae. BMC Biol 11(1):24
Nolt JK, et al.  (2011) PP2A (Cdc)55 is required for multiple events during meiosis I. Cell Cycle 10(9):1420-34
Banuelos MG, et al.  (2010) Genomic analysis of severe hypersensitivity to hygromycin B reveals linkage to vacuolar defects and new vacuolar gene functions in Saccharomyces cerevisiae. Curr Genet 56(2):121-37
Couthouis J, et al.  (2010) The toxicity of an "artificial" amyloid is related to how it interacts with membranes. Prion 4(4):283-91
Tate JJ, et al.  (2010) Distinct phosphatase requirements and GATA factor responses to nitrogen catabolite repression and rapamycin treatment in Saccharomyces cerevisiae. J Biol Chem 285(23):17880-95
McCourt PC, et al.  (2009) Stress-induced Ceramide-activated Protein Phosphatase Can Compensate for Loss of Amphiphysin-like Activity In Saccharomyces cerevisiae and Functions to Reinitiate Endocytosis. J Biol Chem 284(18):11930-41
Tate JJ, et al.  (2009) Rapamycin-induced Gln3 Dephosphorylation Is Insufficient for Nuclear Localization: Sit4 AND PP2A PHOSPHATASES ARE REGULATED AND FUNCTION DIFFERENTLY. J Biol Chem 284(4):2522-34
Li Y, et al.  (2008) Cdc55p-mediated E4orf4 growth inhibition in Saccharomyces cerevisiae is mediated only in part via the catalytic subunit of protein phosphatase 2A. J Virol 82(7):3612-23
Queralt E and Uhlmann F  (2008) Separase cooperates with Zds1 and Zds2 to activate Cdc14 phosphatase in early anaphase. J Cell Biol 182(5):873-83
Hombauer H, et al.  (2007) Generation of active protein phosphatase 2A is coupled to holoenzyme assembly. PLoS Biol 5(6):e155
Pagani MA, et al.  (2007) Disruption of iron homeostasis in Saccharomyces cerevisiae by high zinc levels: a genome-wide study. Mol Microbiol 65(2):521-37
Tang X and Wang Y  (2006) Pds1/Esp1-dependent and -independent sister chromatid separation in mutants defective for protein phosphatase 2A. Proc Natl Acad Sci U S A 103(44):16290-5
Tomas-Cobos L, et al.  (2005) TOR kinase pathway and 14-3-3 proteins regulate glucose-induced expression of HXT1, a yeast low-affinity glucose transporter. Yeast 22(6):471-9
Koren R, et al.  (2004) The scaffolding A/Tpd3 subunit and high phosphatase activity are dispensable for Cdc55 function in the Saccharomyces cerevisiae spindle checkpoint and in cytokinesis. J Biol Chem 279(47):48598-606
Gimeno-Alcaniz JV and Sanz P  (2003) Glucose and type 2A protein phosphatase regulate the interaction between catalytic and regulatory subunits of AMP-activated protein kinase. J Mol Biol 333(1):201-9
Gentry MS and Hallberg RL  (2002) Localization of Saccharomyces cerevisiae protein phosphatase 2A subunits throughout mitotic cell cycle. Mol Biol Cell 13(10):3477-92
Yu S, et al.  (2001) Protein phosphatase 2A: identification in Oryza sativa of the gene encoding the regulatory A subunit. Plant Mol Biol 45(1):107-12
Jiang Y and Broach JR  (1999) Tor proteins and protein phosphatase 2A reciprocally regulate Tap42 in controlling cell growth in yeast. EMBO J 18(10):2782-92
Wang Y and Burke DJ  (1997) Cdc55p, the B-type regulatory subunit of protein phosphatase 2A, has multiple functions in mitosis and is required for the kinetochore/spindle checkpoint in Saccharomyces cerevisiae. Mol Cell Biol 17(2):620-6
Garbers C, et al.  (1996) A mutation in protein phosphatase 2A regulatory subunit A affects auxin transport in Arabidopsis. EMBO J 15(9):2115-24
Nickels JT and Broach JR  (1996) A ceramide-activated protein phosphatase mediates ceramide-induced G1 arrest of Saccharomyces cerevisiae. Genes Dev 10(4):382-94
Barton AB and Kaback DB  (1994) Molecular cloning of chromosome I DNA from Saccharomyces cerevisiae: analysis of the genes in the FUN38-MAK16-SPO7 region. J Bacteriol 176(7):1872-80
van Zyl W, et al.  (1992) Inactivation of the protein phosphatase 2A regulatory subunit A results in morphological and transcriptional defects in Saccharomyces cerevisiae. Mol Cell Biol 12(11):4946-59