TOR1/YJR066W Literature Guide Help

Other names published for TOR1: DRR1, YJR066W

TOR1 - Primary Literature (113)

Results: 91 - 113 of 113 records
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ReferenceOther Genes Addressed
Kunz J, et al.  (2000) HEAT repeats mediate plasma membrane localization of Tor2p in yeast. J Biol Chem 275(47):37011-20
Shamji AF, et al.  (2000) Partitioning the transcriptional program induced by rapamycin among the effectors of the Tor proteins. Curr Biol 10(24):1574-81
Alarcon CM, et al.  (1999) Protein kinase activity and identification of a toxic effector domain of the target of rapamycin TOR proteins in yeast. Mol Biol Cell 10(8):2531-46
Arndt C, et al.  (1999) Secretion of FK506/FK520 and rapamycin by Streptomyces inhibits the growth of competing Saccharomyces cerevisiae and Cryptococcus neoformans. Microbiology 145 ( Pt 8):1989-2000
Beck T and Hall MN  (1999) The TOR signalling pathway controls nuclear localization of nutrient-regulated transcription factors. Nature 402(6762):689-92
Cardenas ME, et al.  (1999) The TOR signaling cascade regulates gene expression in response to nutrients. Genes Dev 13(24):3271-9
Hardwick JS, et al.  (1999) Rapamycin-modulated transcription defines the subset of nutrient-sensitive signaling pathways directly controlled by the Tor proteins. Proc Natl Acad Sci U S A 96(26):14866-70
Jiang Y and Broach JR  (1999) Tor proteins and protein phosphatase 2A reciprocally regulate Tap42 in controlling cell growth in yeast. EMBO J 18(10):2782-92
Powers T and Walter P  (1999) Regulation of ribosome biogenesis by the rapamycin-sensitive TOR-signaling pathway in Saccharomyces cerevisiae. Mol Biol Cell 10(4):987-1000
Noda T and Ohsumi Y  (1998) Tor, a phosphatidylinositol kinase homologue, controls autophagy in yeast. J Biol Chem 273(7):3963-6
Fiorentino DF and Crabtree GR  (1997) Characterization of Saccharomyces cerevisiae dna2 mutants suggests a role for the helicase late in S phase. Mol Biol Cell 8(12):2519-37
Zheng XF and Schreiber SL  (1997) Target of rapamycin proteins and their kinase activities are required for meiosis. Proc Natl Acad Sci U S A 94(7):3070-5
Alarcon CM, et al.  (1996) Mammalian RAFT1 kinase domain provides rapamycin-sensitive TOR function in yeast. Genes Dev 10(3):279-88
Barbet NC, et al.  (1996) TOR controls translation initiation and early G1 progression in yeast. Mol Biol Cell 7(1):25-42
Freeman K and Livi GP  (1996) Missense mutations at the FKBP12-rapamycin-binding site of TOR1. Gene 172(1):143-7
Lorenz MC and Heitman J  (1995) TOR mutations confer rapamycin resistance by preventing interaction with FKBP12-rapamycin. J Biol Chem 270(46):27531-7
Zheng XF, et al.  (1995) TOR kinase domains are required for two distinct functions, only one of which is inhibited by rapamycin. Cell 82(1):121-30
Cafferkey R, et al.  (1994) Yeast TOR (DRR) proteins: amino-acid sequence alignment and identification of structural motifs. Gene 141(1):133-6
Helliwell SB, et al.  (1994) TOR1 and TOR2 are structurally and functionally similar but not identical phosphatidylinositol kinase homologues in yeast. Mol Biol Cell 5(1):105-18
Stan R, et al.  (1994) Interaction between FKBP12-rapamycin and TOR involves a conserved serine residue. J Biol Chem 269(51):32027-30
Cafferkey R, et al.  (1993) Dominant missense mutations in a novel yeast protein related to mammalian phosphatidylinositol 3-kinase and VPS34 abrogate rapamycin cytotoxicity. Mol Cell Biol 13(10):6012-23
Kunz J, et al.  (1993) Target of rapamycin in yeast, TOR2, is an essential phosphatidylinositol kinase homolog required for G1 progression. Cell 73(3):585-96
Heitman J, et al.  (1991) Targets for cell cycle arrest by the immunosuppressant rapamycin in yeast. Science 253(5022):905-9
Results: 91 - 113 of 113 records
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