SAE2/YGL175C Literature Guide Help

Other names published for SAE2: COM1, YGL175C

SAE2 - Omics (22)

ReferenceOther Genes Addressed
Guenole A, et al.  (2013) Dissection of DNA damage responses using multiconditional genetic interaction maps. Mol Cell 49(2):346-58
Debacker K, et al.  (2012) Histone deacetylase complexes promote trinucleotide repeat expansions. PLoS Biol 10(2):e1001257
Douglas AC, et al.  (2012) Functional analysis with a barcoder yeast gene overexpression system. G3 (Bethesda) 2(10):1279-89
Mine-Hattab J and Rothstein R  (2012) Increased chromosome mobility facilitates homology search during recombination.LID - 10.1038/ncb2472 [doi] Nat Cell Biol ()
Psakhye I and Jentsch S  (2012) Protein group modification and synergy in the SUMO pathway as exemplified in DNA repair. Cell 151(4):807-20
Zhang Y, et al.  (2012) Genome-wide screen identifies pathways that govern GAA/TTC repeat fragility and expansions in dividing and nondividing yeast cells. Mol Cell 48(2):254-65
Reid RJ, et al.  (2011) Selective ploidy ablation, a high-throughput plasmid transfer protocol, identifies new genes affecting topoisomerase I-induced DNA damage. Genome Res 21(3):477-86
Casper AM, et al.  (2009) Chromosome aberrations resulting from double-strand DNA breaks at a naturally occurring yeast fragile site composed of inverted ty elements are independent of Mre11p and Sae2p. Genetics 183(2):423-39, 1SI-26SI
Nyswaner KM, et al.  (2008) Chromatin-associated genes protect the yeast genome from ty1 insertional mutagenesis. Genetics 178(1):197-214
Alvaro D, et al.  (2007) Genome-wide analysis of Rad52 foci reveals diverse mechanisms impacting recombination. PLoS Genet 3(12):e228
Buhler C, et al.  (2007) Mapping meiotic single-strand dna reveals a new landscape of DNA double-strand breaks in Saccharomyces cerevisiae. PLoS Biol 5(12):e324
Kitagawa T, et al.  (2007) Genome-Wide Analysis of Cellular Response to Bacterial Genotoxin CdtB in Yeast. Infect Immun 75(3):1393-402
Liao C, et al.  (2007) Genomic Screening in Vivo Reveals the Role Played by Vacuolar H+ ATPase and Cytosolic Acidification in Sensitivity to DNA-Damaging Agents Such as Cisplatin. Mol Pharmacol 71(2):416-25
Sawyer SL and Malik HS  (2006) Positive selection of yeast nonhomologous end-joining genes and a retrotransposon conflict hypothesis. Proc Natl Acad Sci U S A 103(47):17614-9
Deng C, et al.  (2005) Multiple endonucleases function to repair covalent topoisomerase I complexes in Saccharomyces cerevisiae. Genetics 170(2):591-600
Tong AH, et al.  (2004) Global mapping of the yeast genetic interaction network. Science 303(5659):808-13
Game JC, et al.  (2003) Use of a genome-wide approach to identify new genes that control resistance of Saccharomyces cerevisiae to ionizing radiation. Radiat Res 160(1):14-24
Huh WK, et al.  (2003) Global analysis of protein localization in budding yeast. Nature 425(6959):686-91
Birrell GW, et al.  (2002) Transcriptional response of Saccharomyces cerevisiae to DNA-damaging agents does not identify the genes that protect against these agents. Proc Natl Acad Sci U S A 99(13):8778-83
Chang M, et al.  (2002) A genome-wide screen for methyl methanesulfonate-sensitive mutants reveals genes required for S phase progression in the presence of DNA damage. Proc Natl Acad Sci U S A 99(26):16934-9
Birrell GW, et al.  (2001) A genome-wide screen in Saccharomyces cerevisiae for genes affecting UV radiation sensitivity. Proc Natl Acad Sci U S A 98(22):12608-13
Tong AH, et al.  (2001) Systematic genetic analysis with ordered arrays of yeast deletion mutants. Science 294(5550):2364-8