Other names published for MEC1: ESR1, SAD3, RAD31, YBR136W
MEC1 LITERATURE TOPICS
- Curated Literature
- Genetics/Cell Biology
- Nucleic Acid Information
- Gene Product Information
- Related Genes/Proteins
- Research Aids
- Genome-wide Analysis
- Proteome-wide Analysis
- Other Topics
- Additional Information
MEC1 - Omics (56)
| Reference | Other Genes Addressed |
|---|---|
| Foltman M, et al. (2013) Eukaryotic replisome components cooperate to process histones during chromosome replication. Cell Rep 3(3):892-904 | |
| Rodriguez J and Tsukiyama T (2013) ATR-like kinase Mec1 facilitates both chromatin accessibility at DNA replication forks and replication fork progression during replication stress. Genes Dev 27(1):74-86 | |
| Cheung HC, et al. (2012) An S/T-Q cluster domain census unveils new putative targets under Tel1/Mec1 control. BMC Genomics 13(1):664 | |
| Davidson MB, et al. (2012) Endogenous DNA replication stress results in expansion of dNTP pools and a mutator phenotype. EMBO J 31(4):895-907 | |
| De Piccoli G, et al. (2012) Replisome stability at defective DNA replication forks is independent of S phase checkpoint kinases. Mol Cell 45(5):696-704 | |
| Lai AC, et al. (2012) Predicting kinase substrates using conservation of local motif density. Bioinformatics 28(7):962-9 | |
| Liang D, et al. (2012) Histone dosage regulates DNA damage sensitivity in a checkpoint-independent manner by the homologous recombination pathway. Nucleic Acids Res 40(19):9604-20 | |
| Psakhye I and Jentsch S (2012) Protein group modification and synergy in the SUMO pathway as exemplified in DNA repair. Cell 151(4):807-20 | |
| Tittel-Elmer M, et al. (2012) Cohesin association to replication sites depends on rad50 and promotes fork restart. Mol Cell 48(1):98-108 | |
| Tkach JM, et al. (2012) Dissecting DNA damage response pathways by analysing protein localization and abundance changes during DNA replication stress. Nat Cell Biol 14(9):966-76 | |
| Feng W, et al. (2011) Replication stress-induced chromosome breakage is correlated with replication fork progression and is preceded by single-stranded DNA formation. G3 (Bethesda) 1(5):327-35 | |
| Kitada T, et al. (2011) gammaH2A is a component of yeast heterochromatin required for telomere elongation. Cell Cycle 10(2):293-300 | |
| Kubota T, et al. (2011) Quantitative proteomic analysis of chromatin reveals that Ctf18 acts in the DNA replication checkpoint. Mol Cell Proteomics 10(7):M110.005561 | |
| Leon Ortiz AM, et al. (2011) Srs2 overexpression reveals a helicase-independent role at replication forks that requires diverse cell functions. DNA Repair (Amst) 10(5):506-17 | |
| Reha-Krantz LJ, et al. (2011) Drug-sensitive DNA polymerase d reveals a role for mismatch repair in checkpoint activation in yeast. Genetics 189(4):1211-24 | |
| Bandyopadhyay S, et al. (2010) Rewiring of genetic networks in response to DNA damage. Science 330(6009):1385-9 | |
| Chen SH, et al. (2010) A proteome-wide analysis of kinase-substrate network in the DNA damage response. J Biol Chem 285(17):12803-12 | |
| Crabbe L, et al. (2010) Analysis of replication profiles reveals key role of RFC-Ctf18 in yeast replication stress response. Nat Struct Mol Biol 17(11):1391-1397 | |
| Jarosz DF and Lindquist S (2010) Hsp90 and environmental stress transform the adaptive value of natural genetic variation. Science 330(6012):1820-4 | |
| Lopez-Mosqueda J, et al. (2010) Damage-induced phosphorylation of Sld3 is important to block late origin firing. Nature 467(7314):479-83 | |
| On T, et al. (2010) The evolutionary landscape of the chromatin modification machinery reveals lineage specific gains, expansions, and losses. Proteins 78(9):2075-89 | |
| Rogers C, et al. (2010) Connecting mutations of the RNA polymerase II C-terminal domain to complex phenotypic changes using combined gene expression and network analyses. PLoS One 5(6):e11386 | |
| Szilard RK, et al. (2010) Systematic identification of fragile sites via genome-wide location analysis of gamma-H2AX. Nat Struct Mol Biol 17(3):299-305 | |
| Theis JF, et al. (2010) The DNA Damage Response Pathway Contributes to the Stability of Chromosome III Derivatives Lacking Efficient Replicators. PLoS Genet 6(12):e1001227 | |
| Karhumaa K, et al. (2009) Proteome analysis of the xylose-fermenting mutant yeast strain TMB 3400. Yeast 26(7):371-82 | |
| Molin C, et al. (2009) mRNA stability changes precede changes in steady-state mRNA amounts during hyperosmotic stress. RNA 15(4):600-14 | |
| Ohuchi T, et al. (2009) Accumulation of sumoylated Rad52 in checkpoint mutants perturbed in DNA replication. DNA Repair (Amst) 8(6):690-6 | |
| Pennaneach V and Kolodner RD (2009) Stabilization of dicentric translocations through secondary rearrangements mediated by multiple mechanisms in S. cerevisiae. PLoS One 4(7):e6389 | |
| Albuquerque CP, et al. (2008) A multidimensional chromatography technology for in-depth phosphoproteome analysis. Mol Cell Proteomics 7(7):1389-96 | |
| Dixon SJ, et al. (2008) Significant conservation of synthetic lethal genetic interaction networks between distantly related eukaryotes. Proc Natl Acad Sci U S A 105(43):16653-8 |




