Other names published for RAD14: YMR201C
RAD14 LITERATURE TOPICS
- Curated Literature
- Genetics/Cell Biology
- Cell Cycle Phase Involved
- Cellular Location
- Function/Process
- Genetic Interactions
- Mutants/Phenotypes
- Regulation of
- Nucleic Acid Information
- Gene Product Information
- Related Genes/Proteins
- Research Aids
- Genome-wide Analysis
- Proteome-wide Analysis
- Additional Information
RAD14 - Mutants/Phenotypes (85)
| Reference | Other Genes Addressed |
|---|---|
| Cheung-Ong K, et al. (2012) Comparative chemogenomics to examine the mechanism of action of dna-targeted platinum-acridine anticancer agents. ACS Chem Biol 7(11):1892-901 | |
| Collura A, et al. (2012) Abasic sites linked to dUTP incorporation in DNA are a major cause of spontaneous mutations in absence of base excision repair and Rad17-Mec3-Ddc1 (9-1-1) DNA damage checkpoint clamp in Saccharomyces cerevisiae. DNA Repair (Amst) 11(3):294-303 | |
| Grogan D and Jinks-Robertson S (2012) Formaldehyde-induced mutagenesis in Saccharomyces cerevisiae: molecular properties and the roles of repair and bypass systems. Mutat Res 731(1-2):92-8 | |
| Haruta N, et al. (2012) Chronic low-dose ultraviolet-induced mutagenesis in nucleotide excision repair-deficient cells. Nucleic Acids Res 40(17):8406-15 | |
| Lazzaro F, et al. (2012) RNase H and postreplication repair protect cells from ribonucleotides incorporated in DNA. Mol Cell 45(1):99-110 | |
| Stone JE, et al. (2012) DNA polymerase zeta generates clustered mutations during bypass of endogenous DNA lesions in Saccharomyces cerevisiae. Environ Mol Mutagen 53(9):777-86 | |
| Gangavarapu V, et al. (2011) Requirement of replication checkpoint protein kinases mec1/rad53 for postreplication repair in yeast.LID - 10.1128/mBio.00079-11 [doi]LID - e00079-11 [pii] MBio 2(3) | |
| Kim N, et al. (2011) The dCMP transferase activity of yeast Rev1 is biologically relevant during the bypass of endogenously generated AP sites. DNA Repair (Amst) 10(12):1262-71 | |
| Mardiros A, et al. (2011) Rad10-YFP focus induction in response to UV depends on RAD14 in yeast. Acta Histochem 113(4):409-15 | |
| Silver HR, et al. (2011) A role for SUMO in nucleotide excision repair. DNA Repair (Amst) 10(12):1243-51 | |
| Svensson JP, et al. (2011) Genomic phenotyping of the essential and non-essential yeast genome detects novel pathways for alkylation resistance. BMC Syst Biol 5(1):157 | |
| Wiltrout ME and Walker GC (2011) The DNA Polymerase Activity of Saccharomyces cerevisiae Rev1 is Biologically Significant. Genetics 187(1):21-35 | |
| Heidenreich E, et al. (2010) A mutation-promotive role of nucleotide excision repair in cell cycle-arrested cell populations following UV irradiation. DNA Repair (Amst) 9(1):96-100 | |
| Kim N and Jinks-Robertson S (2010) Abasic sites in the transcribed strand of yeast DNA are removed by transcription-coupled nucleotide excision repair. Mol Cell Biol 30(13):3206-15 | |
| Minca EC and Kowalski D (2010) Multiple Rad5 activities mediate sister chromatid recombination to bypass DNA damage at stalled replication forks. Mol Cell 38(5):649-61 | |
| Northam MR, et al. (2010) Participation of DNA polymerase {zeta} in replication of undamaged DNA in Saccharomyces cerevisiae. Genetics 184(1):27-42 | |
| Serrentino ME, et al. (2010) Photosensitization induced by the antibacterial fluoroquinolone Rufloxacin leads to mutagenesis in yeast. Mutat Res 692(1-2):34-41 | |
| Toussaint M, et al. (2010) Differential participation of homologous recombination and nucleotide excision repair in yeast survival to ultraviolet light radiation. Mutat Res 698(1-2):52-59 | |
| Hishida T, et al. (2009) RAD6-RAD18-RAD5-pathway-dependent tolerance to chronic low-dose ultraviolet light. Nature 457(7229):612-5 | |
| Lehner K and Jinks-Robertson S (2009) The mismatch repair system promotes DNA polymerase zeta-dependent translesion synthesis in yeast. Proc Natl Acad Sci U S A 106(14):5749-54 | |
| Pages V, et al. (2009) Role of DNA damage-induced replication checkpoint in promoting lesion bypass by translesion synthesis in yeast. Genes Dev 23(12):1438-49 | |
| de Graaf B, et al. (2009) Cellular pathways for DNA repair and damage tolerance of formaldehyde-induced DNA-protein crosslinks. DNA Repair (Amst) 8(10):1207-14 | |
| Matsufuji Y, et al. (2008) Acetaldehyde tolerance in Saccharomyces cerevisiae involves the pentose phosphate pathway and oleic acid biosynthesis. Yeast 25(11):825-33 | |
| Monti P, et al. (2008) Rev1 and Polzeta influence toxicity and mutagenicity of Me-lex, a sequence selective N3-adenine methylating agent. DNA Repair (Amst) 7(3):431-8 | |
| Serero A, et al. (2008) Yeast genes involved in cadmium tolerance: Identification of DNA replication as a target of cadmium toxicity. DNA Repair (Amst) 7(8):1262-75 | |
| Yu L, et al. (2008) Chemical-genetic profiling of imidazo[1,2-a]pyridines and -pyrimidines reveals target pathways conserved between yeast and human cells. PLoS Genet 4(11):e1000284 | |
| Abdulovic AL, et al. (2007) Identification of a strand-related bias in the PCNA-mediated bypass of spontaneous lesions by yeast Poleta. DNA Repair (Amst) 6(9):1307-18 | |
| Liao C, et al. (2007) Genomic Screening in Vivo Reveals the Role Played by Vacuolar H+ ATPase and Cytosolic Acidification in Sensitivity to DNA-Damaging Agents Such as Cisplatin. Mol Pharmacol 71(2):416-25 | |
| Boiteux S and Guillet M (2006) Use of yeast for detection of endogenous abasic lesions, their source, and their repair. Methods Enzymol 408:79-91 | |
| Lopes M, et al. (2006) Multiple mechanisms control chromosome integrity after replication fork uncoupling and restart at irreparable UV lesions. Mol Cell 21(1):15-27 |



