Other names published for PFK1: 6-phosphofructokinase subunit alpha, YGR240C
PFK1 LITERATURE TOPICS
- Curated Literature
- Genetics/Cell Biology
- Nucleic Acid Information
- Gene Product Information
- Related Genes/Proteins
- Research Aids
- Genome-wide Analysis
- Proteome-wide Analysis
- Other Topics
- Additional Information
PFK1 - Mutants/Phenotypes (40)
| Reference | Other Genes Addressed |
|---|---|
| Martinez-Costa OH, et al. (2012) Distinct functional roles of the two terminal halves of eukaryotic phosphofructokinase. Biochem J 445(2):213-8 | |
| Williamson T, et al. (2012) Exploring the genetic control of glycolytic oscillations in Saccharomyces Cerevisiae. BMC Syst Biol 6(1):108 | |
| Kennedy MA, et al. (2011) Srf1 is a novel regulator of phospholipase d activity and is essential to buffer the toxic effects of c16:0 platelet activating factor. PLoS Genet 7(2):e1001299 | |
| Laporte D, et al. (2011) Metabolic status rather than cell cycle signals control quiescence entry and exit. J Cell Biol 192(6):949-57 | |
| Matsufuji Y, et al. (2010) Transcription factor Stb5p is essential for acetaldehyde tolerance in Saccharomyces cerevisiae. J Basic Microbiol 50(5):494-8 | |
| Mira NP, et al. (2010) Genome-wide identification of Saccharomyces cerevisiae genes required for tolerance to acetic acid. Microb Cell Fact 9(1):79 | |
| Ruotolo R, et al. (2010) Chemogenomic profiling of the cellular effects associated with histone H3 acetylation impairment by a quinoline-derived compound. Genomics 96(5):272-80 | |
| Teixeira MC, et al. (2010) Identification of genes required for maximal tolerance to high-glucose concentrations, as those present in industrial alcoholic fermentation media, through a chemogenomics approach. OMICS 14(2):201-10 | |
| Matsufuji Y, et al. (2008) Acetaldehyde tolerance in Saccharomyces cerevisiae involves the pentose phosphate pathway and oleic acid biosynthesis. Yeast 25(11):825-33 | |
| Su Y, et al. (2008) Human H+ATPase a4 subunit mutations causing renal tubular acidosis reveal a role for interaction with phosphofructokinase-1. Am J Physiol Renal Physiol 295(4):F950-8 | |
| Bundy JG, et al. (2007) Evaluation of predicted network modules in yeast metabolism using NMR-based metabolite profiling. Genome Res 17(4):510-9 | |
| Freimoser FM, et al. (2006) Systematic screening of polyphosphate (poly P) levels in yeast mutant cells reveals strong interdependence with primary metabolism. Genome Biol 7(11):R109 | |
| Dong L and Xu CW (2004) Carbohydrates induce mono-ubiquitination of H2B in yeast. J Biol Chem 279(3):1577-80 | |
| Wilson WA, et al. (2004) Increased glycogen storage in yeast results in less branched glycogen. Biochem Biophys Res Commun 320(2):416-23 | |
| Newcomb LL, et al. (2003) Glucose regulation of Saccharomyces cerevisiae cell cycle genes. Eukaryot Cell 2(1):143-9 | |
| Edelmann A, et al. (2002) C-terminal modification of 6-phosphofructo-1-kinase from Saccharomyces cerevisiae and its influence on enzyme structure and activity. Biochem Biophys Res Commun 295(4):992-9 | |
| Steinmetz LM, et al. (2002) Systematic screen for human disease genes in yeast. Nat Genet 31(4):400-4 | |
| Pearce AK, et al. (2001) Genetic manipulation of 6-phosphofructo-1-kinase and fructose 2,6-bisphosphate levels affects the extent to which benzoic acid inhibits the growth of Saccharomyces cerevisiae. Microbiology 147(Pt 2):403-10 | |
| Pearce AK, et al. (2001) Pyruvate kinase (Pyk1) levels influence both the rate and direction of carbon flux in yeast under fermentative conditions. Microbiology 147(Pt 2):391-401 | |
| Rodicio R, et al. (2000) Single point mutations in either gene encoding the subunits of the heterooctameric yeast phosphofructokinase abolish allosteric inhibition by ATP. J Biol Chem 275(52):40952-60 | |
| van Hoek P, et al. (2000) Regulation of fermentative capacity and levels of glycolytic enzymes in chemostat cultures of Saccharomyces cerevisiae. Enzyme Microb Technol 26(9-10):724-736 | |
| Cheng L, et al. (1999) Weak organic acid treatment causes a trehalose accumulation in low-pH cultures of Saccharomyces cerevisiae, not displayed by the more preservative-resistant Zygosaccharomyces bailii. FEMS Microbiol Lett 170(1):89-95 | |
| Kirchberger J, et al. (1999) A single point mutation leads to an instability of the hetero-octameric structure of yeast phosphofructokinase. Biochem J 341 ( Pt 1)():15-23 | |
| Klinder A, et al. (1998) Assembly of phosphofructokinase-1 from Saccharomyces cerevisiae in extracts of single-deletion mutants. Yeast 14(4):323-34 | |
| Heinisch JJ, et al. (1996) A yeast phosphofructokinase insensitive to the allosteric activator fructose 2,6-bisphosphate. Glycolysis/metabolic regulation/allosteric control. J Biol Chem 271(27):15928-33 | |
| Holyoak CD, et al. (1996) Activity of the plasma membrane H(+)-ATPase and optimal glycolytic flux are required for rapid adaptation and growth of Saccharomyces cerevisiae in the presence of the weak-acid preservative sorbic acid. Appl Environ Microbiol 62(9):3158-64 | |
| Muller S, et al. (1996) A two-hybrid system analysis shows interactions between 6-phosphofructo-1-kinase and 6-phosphofructo-2-kinase but not between other glycolytic enzymes of the yeast Saccharomyces cerevisiae. Eur J Biochem 236(2):626-31 | |
| Arvanitidis A and Heinisch JJ (1994) Studies on the function of yeast phosphofructokinase subunits by in vitro mutagenesis. J Biol Chem 269(12):8911-8 | |
| Boles E, et al. (1993) Different signals control the activation of glycolysis in the yeast Saccharomyces cerevisiae. Yeast 9(7):761-70 | |
| Heinisch J, et al. (1993) Molecular genetics of phosphofructokinase in the yeast Kluyveromyces lactis. Mol Microbiol 8(3):559-70 |




