MEC3/YLR288C Literature Guide Help

Other names published for MEC3: PIP3, PSO9, YLR288C

MEC3 - Mutants/Phenotypes (59)

ReferenceOther Genes Addressed
Becerra SC, et al.  (2012) Reversibility of replicative senescence in Saccharomyces cerevisiae: effect of homologous recombination and cell cycle checkpoints. DNA Repair (Amst) 11(1):35-45
Costelloe T, et al.  (2012) The yeast Fun30 and human SMARCAD1 chromatin remodellers promote DNA end resection. Nature 489(7417):581-4
Paschini M, et al.  (2012) A naturally thermolabile activity compromises genetic analysis of telomere function in Saccharomyces cerevisiae. Genetics 191(1):79-93
Yu S, et al.  (2012) Compromised cellular responses to DNA damage accelerate chronological aging by incurring cell wall fragility in Saccharomyces cerevisiae. Mol Biol Rep 39(4):3573-83
Ambroset C, et al.  (2011) Deciphering the molecular basis of wine yeast fermentation traits using a combined genetic and genomic approach. G3 (Bethesda) 1(4):263-81
Germann SM, et al.  (2011) Dpb11/TopBP1 plays distinct roles in DNA replication, checkpoint response and homologous recombination. DNA Repair (Amst) 10(2):210-24
Reha-Krantz LJ, et al.  (2011) Drug-sensitive DNA polymerase d reveals a role for mismatch repair in checkpoint activation in yeast. Genetics 189(4):1211-24
Reid RJ, et al.  (2011) Selective ploidy ablation, a high-throughput plasmid transfer protocol, identifies new genes affecting topoisomerase I-induced DNA damage. Genome Res 21(3):477-86
Schmidt KH, et al.  (2010) Defects in DNA lesion bypass lead to spontaneous chromosomal rearrangements and increased cell death. Eukaryot Cell 9(2):315-24
Schmidt KH, et al.  (2010) Formation of complex and unstable chromosomal translocations in yeast.LID - e12007 [pii] PLoS One 5(8)
Gomez-Gonzalez B, et al.  (2009) The S-phase checkpoint is required to respond to R-loops accumulated in THO mutants. Mol Cell Biol 29(19):5203-13
Pages V, et al.  (2009) Role of DNA damage-induced replication checkpoint in promoting lesion bypass by translesion synthesis in yeast. Genes Dev 23(12):1438-49
Barlow JH, et al.  (2008) Differential regulation of the cellular response to DNA double-strand breaks in G1. Mol Cell 30(1):73-85
Cardone JM, et al.  (2008) DNA repair by polymerase delta in Saccharomyces cerevisiae is not controlled by the proliferating cell nuclear antigen-like Rad17/Mec3/Ddc1 complex. Genet Mol Res 7(1):127-32
Razidlo DF and Lahue RS  (2008) Mrc1, Tof1 and Csm3 inhibit CAG.CTG repeat instability by at least two mechanisms. DNA Repair (Amst) 7(4):633-40
Seitomer E, et al.  (2008) Analysis of Saccharomyces cerevisiae null allele strains identifies a larger role for DNA damage versus oxidative stress pathways in growth inhibition by selenium. Mol Nutr Food Res 52(11):1305-15
Kitagawa T, et al.  (2007) Genome-Wide Analysis of Cellular Response to Bacterial Genotoxin CdtB in Yeast. Infect Immun 75(3):1393-402
Cardone JM, et al.  (2006) Psoralen-sensitive mutant pso9-1 of Saccharomyces cerevisiae contains a mutant allele of the DNA damage checkpoint gene MEC3. DNA Repair (Amst) 5(2):163-71
Ogiwara H, et al.  (2006) Dpb11, the budding yeast homolog of TopBP1, functions with the checkpoint clamp in recombination repair. Nucleic Acids Res 34(11):3389-98
Schmidt KH and Kolodner RD  (2006) Suppression of spontaneous genome rearrangements in yeast DNA helicase mutants. Proc Natl Acad Sci U S A 103(48):18196-201
Schmidt KH, et al.  (2006) Control of translocations between highly diverged genes by Sgs1, the Saccharomyces cerevisiae homolog of the Bloom's syndrome protein. Mol Cell Biol 26(14):5406-20
Grandin N, et al.  (2005) Activation of Mrc1, a mediator of the replication checkpoint, by telomere erosion. Biol Cell 97(10):799-814
Huang ME and Kolodner RD  (2005) A biological network in Saccharomyces cerevisiae prevents the deleterious effects of endogenous oxidative DNA damage. Mol Cell 17(5):709-20
Lee W, et al.  (2005) Genome-wide requirements for resistance to functionally distinct DNA-damaging agents. PLoS Genet 1(2):e24
Majka J and Burgers PM  (2005) Function of Rad17/Mec3/Ddc1 and its partial complexes in the DNA damage checkpoint. DNA Repair (Amst) 4(10):1189-94
Putnam CD, et al.  (2005) Saccharomyces cerevisiae as a model system to define the chromosomal instability phenotype. Mol Cell Biol 25(16):7226-38
Sabbioneda S, et al.  (2005) The 9-1-1 checkpoint clamp physically interacts with polzeta and is partially required for spontaneous polzeta-dependent mutagenesis in Saccharomyces cerevisiae. J Biol Chem 280(46):38657-65
Baroni E, et al.  (2004) The functions of budding yeast Sae2 in the DNA damage response require Mec1- and Tel1-dependent phosphorylation. Mol Cell Biol 24(10):4151-65
Jia X, et al.  (2004) Mec1 and Rad53 inhibit formation of single-stranded DNA at telomeres of Saccharomyces cerevisiae cdc13-1 mutants. Genetics 166(2):753-64
Lin J, et al.  (2004) Mutant telomere sequences lead to impaired chromosome separation and a unique checkpoint response. Mol Biol Cell 15(4):1623-34