TSA1/YML028W Literature Guide Help

Other names published for TSA1: ZRG14, TPX1, cTPxI, YML028W

TSA1 - Mutants/Phenotypes (75)

ReferenceOther Genes Addressed
Tachibana T, et al.  (2009) A Major Peroxiredoxin-induced Activation of Yap1 Transcription Factor Is Mediated by Reduction-sensitive Disulfide Bonds and Reveals a Low Level of Transcriptional Activation. J Biol Chem 284(7):4464-72
Tang HM, et al.  (2009) Loss of Yeast Peroxiredoxin Tsa1p Induces Genome Instability through Activation of the DNA Damage Checkpoint and Elevation of dNTP Levels. PLoS Genet 5(10):e1000697
Wu CY, et al.  (2009) Cytosolic superoxide dismutase (SOD1) is critical for tolerating the oxidative stress of zinc deficiency in yeast. PLoS One 4(9):e7061
Degtyareva NP, et al.  (2008) Chronic oxidative DNA damage due to DNA repair defects causes chromosomal instability in Saccharomyces cerevisiae. Mol Cell Biol 28(17):5432-45
Iraqui I, et al.  (2008) Human peroxiredoxin PrxI is an orthologue of yeast Tsa1, capable of suppressing genome instability in Saccharomyces cerevisiae. Cancer Res 68(4):1055-63
Mroczek S and Kufel J  (2008) Apoptotic signals induce specific degradation of ribosomal RNA in yeast. Nucleic Acids Res 36(9):2874-88
Roussel X, et al.  (2008) Evidence for the formation of a covalent thiosulfinate intermediate with peroxiredoxin in the catalytic mechanism of sulfiredoxin. J Biol Chem 283(33):22371-82
Seitomer E, et al.  (2008) Analysis of Saccharomyces cerevisiae null allele strains identifies a larger role for DNA damage versus oxidative stress pathways in growth inhibition by selenium. Mol Nutr Food Res 52(11):1305-15
Trotter EW, et al.  (2008) The yeast Tsa1 peroxiredoxin is a ribosome-associated antioxidant. Biochem J 412(1):73-80
Zadrag R, et al.  (2008) Is the yeast a relevant model for aging of multicellular organisms? An insight from the total lifespan of Saccharomyces cerevisiae. Curr Aging Sci 1(3):159-65
Doostzadeh J, et al.  (2007) Chemical genomic profiling for identifying intracellular targets of toxicants producing Parkinson's disease. Toxicol Sci 95(1):182-7
Lopez-Mirabal HR, et al.  (2007) Cytoplasmic glutathione redox status determines survival upon exposure to the thiol-oxidant 4,4'-dipyridyl disulfide. FEMS Yeast Res 7(3):391-403
Milgrom E, et al.  (2007) Loss of vacuolar proton-translocating ATPase activity in yeast results in chronic oxidative stress. J Biol Chem 282(10):7125-36
Ragu S, et al.  (2007) Oxygen metabolism and reactive oxygen species cause chromosomal rearrangements and cell death. Proc Natl Acad Sci U S A 104(23):9747-52
Wu CY, et al.  (2007) Regulation of the yeast TSA1 peroxiredoxin by ZAP1 is an adaptive response to the oxidative stress of zinc deficiency. J Biol Chem 282(4):2184-95
de Oliveira MA, et al.  (2007) Crystallization and preliminary X-ray analysis of a decameric form of cytosolic thioredoxin peroxidase 1 (Tsa1), C47S mutant, from Saccharomyces cerevisiae. Acta Crystallogr Sect F Struct Biol Cryst Commun 63(Pt 8):665-8
Brombacher K, et al.  (2006) The role of Yap1p and Skn7p-mediated oxidative stress response in the defence of Saccharomyces cerevisiae against singlet oxygen. Yeast 23(10):741-50
Demasi AP, et al.  (2006) Yeast oxidative stress response. Influences of cytosolic thioredoxin peroxidase I and of the mitochondrial functional state. FEBS J 273(4):805-16
Kim JH, et al.  (2006) Controlling food-contaminating fungi by targeting their antioxidative stress-response system with natural phenolic compounds. Appl Microbiol Biotechnol 70(6):735-9
Rand JD and Grant CM  (2006) The thioredoxin system protects ribosomes against stress-induced aggregation. Mol Biol Cell 17(1):387-401
Huang ME and Kolodner RD  (2005) A biological network in Saccharomyces cerevisiae prevents the deleterious effects of endogenous oxidative DNA damage. Mol Cell 17(5):709-20
Okazaki S, et al.  (2005) Peroxiredoxin-mediated redox regulation of the nuclear localization of Yap1, a transcription factor in budding yeast. Antioxid Redox Signal 7(3-4):327-34
Kim JH, et al.  (2004) Secondary metabolites of the grapevine pathogen Eutypa lata inhibit mitochondrial respiration, based on a model bioassay using the yeast Saccharomyces cerevisiae. Curr Microbiol 49(4):282-7
Monteiro G, et al.  (2004) Glutathione and thioredoxin peroxidases mediate susceptibility of yeast mitochondria to Ca(2+)-induced damage. Arch Biochem Biophys 425(1):14-24
Munhoz DC and Netto LE  (2004) Cytosolic thioredoxin peroxidase I and II are important defenses of yeast against organic hydroperoxide insult: catalases and peroxiredoxins cooperate in the decomposition of H2O2 by yeast. J Biol Chem 279(34):35219-27
Smith S, et al.  (2004) Mutator genes for suppression of gross chromosomal rearrangements identified by a genome-wide screening in Saccharomyces cerevisiae. Proc Natl Acad Sci U S A 101(24):9039-44
Tucker CL and Fields S  (2004) Quantitative genome-wide analysis of yeast deletion strain sensitivities to oxidative and chemical stress. Comp Funct Genomics 5(3):216-24
Wong CM, et al.  (2004) Peroxiredoxin-null yeast cells are hypersensitive to oxidative stress and are genomically unstable. J Biol Chem 279(22):23207-13
Huang ME, et al.  (2003) A genomewide screen in Saccharomyces cerevisiae for genes that suppress the accumulation of mutations. Proc Natl Acad Sci U S A 100(20):11529-34
Sakamoto A, et al.  (2003) Functional complementation in yeast reveals a protective role of chloroplast 2-Cys peroxiredoxin against reactive nitrogen species. Plant J 33(5):841-51