FBP1/YLR377C Literature Guide Help

Other names published for FBP1: ACN8, fructose 1,6-bisphosphate 1-phosphatase, YLR377C

FBP1 - Genomic expression study (22)

ReferenceOther Genes Addressed
Dikicioglu D, et al.  (2012) Short- and long-term dynamic responses of the metabolic network and gene expression in yeast to a transient change in the nutrient environment. Mol Biosyst 8(6):1760-74
Duenas-Sanchez R, et al.  (2012) Transcriptional regulation of fermentative and respiratory metabolism in Saccharomyces cerevisiae industrial bakers' strains. FEMS Yeast Res 12(6):625-36
Manikova D, et al.  (2012) Selenium toxicity toward yeast as assessed by microarray analysis and deletion mutant library screen: a role for DNA repair. Chem Res Toxicol 25(8):1598-608
Arribere JA, et al.  (2011) Reconsidering Movement of Eukaryotic mRNAs between Polysomes and P Bodies. Mol Cell 44(5):745-58
Infante JJ, et al.  (2011) Activator-independent transcription of Snf1-dependent genes in mutants lacking histone tails. Mol Microbiol 80(2):407-22
Otero JM, et al.  (2010) Whole genome sequencing of Saccharomyces cerevisiae: from genotype to phenotype for improved metabolic engineering applications. BMC Genomics 11():723
Abe H, et al.  (2009) Upregulation of genes involved in gluconeogenesis and the glyoxylate cycle suppressed the drug sensitivity of an N-glycan-deficient Saccharomyces cerevisiae mutant. Biosci Biotechnol Biochem 73(6):1398-403
Roberts GG 3rd and Hudson AP  (2009) Rsf1p is required for an efficient metabolic shift from fermentative to glycerol-based respiratory growth in S. cerevisiae. Yeast 26(2):95-110
Young ET, et al.  (2009) Snf1-independent, glucose-resistant transcription of Adr1-dependent genes in a mediator mutant of Saccharomyces cerevisiae. Mol Microbiol 74(2):364-83
Bonander N, et al.  (2008) Transcriptome analysis of a respiratory Saccharomycescerevisiae strain suggests the expression of its phenotype is glucose insensitive and predominantly controlled by Hap4, Cat8 and Mig1. BMC Genomics 9:365
Jin C, et al.  (2007) SIT4 regulation of Mig1p-mediated catabolite repression in Saccharomyces cerevisiae. FEBS Lett 581(29):5658-63
Vemuri GN, et al.  (2007) Increasing NADH oxidation reduces overflow metabolism in Saccharomyces cerevisiae. Proc Natl Acad Sci U S A 104(7):2402-7
Tanaka F, et al.  (2006) Functional genomic analysis of commercial baker's yeast during initial stages of model dough-fermentation. Food Microbiol 23(8):717-28
Zhu J, et al.  (2006) A Bayesian Network Driven Approach to Model the Transcriptional Response to Nitric Oxide in Saccharomyces cerevisiae. PLoS ONE 1:e94
Andalis AA, et al.  (2004) Defects arising from whole-genome duplications in Saccharomyces cerevisiae. Genetics 167(3):1109-21
Banerjee D, et al.  (2004) Genome-wide expression profile of steroid response in Saccharomyces cerevisiae. Biochem Biophys Res Commun 317(2):406-13
Daran-Lapujade P, et al.  (2004) Role of transcriptional regulation in controlling fluxes in central carbon metabolism of Saccharomyces cerevisiae. A chemostat culture study. J Biol Chem 279(10):9125-38
Parveen M, et al.  (2004) Response of Saccharomyces cerevisiae to a monoterpene: evaluation of antifungal potential by DNA microarray analysis. J Antimicrob Chemother 54(1):46-55
Boer VM, et al.  (2003) The genome-wide transcriptional responses of Saccharomyces cerevisiae grown on glucose in aerobic chemostat cultures limited for carbon, nitrogen, phosphorus, or sulfur. J Biol Chem 278(5):3265-74
Bro C, et al.  (2003) Transcriptional, proteomic, and metabolic responses to lithium in galactose-grown yeast cells. J Biol Chem 278(34):32141-9
Haurie V, et al.  (2001) The transcriptional activator Cat8p provides a major contribution to the reprogramming of carbon metabolism during the diauxic shift in Saccharomyces cerevisiae. J Biol Chem 276(1):76-85
Schaus SE, et al.  (2001) Gene transcription analysis of Saccharomyces cerevisiae exposed to neocarzinostatin protein-chromophore complex reveals evidence of DNA damage, a potential mechanism of resistance, and consequences of prolonged exposure. Proc Natl Acad Sci U S A 98(20):11075-80