MED2/YDL005C Literature Guide Help

Other names published for MED2: YDL005C

MED2 - Genomic expression study (13)

ReferenceOther Genes Addressed
Miller C, et al.  (2012) Mediator phosphorylation prevents stress response transcription during non-stress conditions. J Biol Chem 287(53):44017-26
Becerra M, et al.  (2011) Comparative transcriptome analysis of yeast strains carrying slt2, rlm1, and pop2 deletions. Genome 54(2):99-109
Toesca I, et al.  (2011) Cryptic transcription mediates repression of subtelomeric metal homeostasis genes. PLoS Genet 7(6):e1002163
Rodriguez-Gil A, et al.  (2010) The distribution of active RNA polymerase II along the transcribed region is gene-specific and controlled by elongation factors. Nucleic Acids Res 38(14):4651-64
Chen AK, et al.  (2009) Response of Saccharomyces cerevisiae to stress-free acidification. J Microbiol 47(1):1-8
Gaillard H, et al.  (2009) Genome-wide analysis of factors affecting transcription elongation and DNA repair: a new role for PAF and Ccr4-not in transcription-coupled repair. PLoS Genet 5(2):e1000364
Koschubs T, et al.  (2009) Identification, structure, and functional requirement of the Mediator submodule Med7N/31. EMBO J 28(1):69-80
Young ET, et al.  (2009) Snf1-independent, glucose-resistant transcription of Adr1-dependent genes in a mediator mutant of Saccharomyces cerevisiae. Mol Microbiol 74(2):364-83
Fong CS, et al.  (2008) Oxidant-induced cell-cycle delay in Saccharomyces cerevisiae: the involvement of the SWI6 transcription factor. FEMS Yeast Res 8(3):386-99
Beve J, et al.  (2005) The structural and functional role of Med5 in the yeast Mediator tail module. J Biol Chem 280(50):41366-72
van de Peppel J, et al.  (2005) Mediator expression profiling epistasis reveals a signal transduction pathway with antagonistic submodules and highly specific downstream targets. Mol Cell 19(4):511-22
Han SJ, et al.  (2001) Med9/Cse2 and Gal11 modules are required for transcriptional repression of distinct group of genes. J Biol Chem 276(40):37020-6
Myers LC, et al.  (1999) Mediator protein mutations that selectively abolish activated transcription. Proc Natl Acad Sci U S A 96(1):67-72