Other names published for SAE2: COM1, YGL175C
SAE2 LITERATURE TOPICS
- Curated Literature
- Genetics/Cell Biology
- Cell Cycle Phase Involved
- Cellular Location
- Function/Process
- Genetic Interactions
- Mutants/Phenotypes
- Regulation of
- Regulatory Role
- Nucleic Acid Information
- Gene Product Information
- Related Genes/Proteins
- Research Aids
- Genome-wide Analysis
- Proteome-wide Analysis
- Other Topics
- Additional Information
SAE2 - Genetic Interactions (36)
| Reference | Other Genes Addressed |
|---|---|
| Costelloe T, et al. (2012) The yeast Fun30 and human SMARCAD1 chromatin remodellers promote DNA end resection. Nature 489(7417):581-4 | |
| Debacker K, et al. (2012) Histone deacetylase complexes promote trinucleotide repeat expansions. PLoS Biol 10(2):e1001257 | |
| Douglas AC, et al. (2012) Functional analysis with a barcoder yeast gene overexpression system. G3 (Bethesda) 2(10):1279-89 | |
| Farmer S, et al. (2012) Budding yeast pch2, a widely conserved meiotic protein, is involved in the initiation of meiotic recombination. PLoS One 7(6):e39724 | |
| Foster SS, et al. (2011) Functional interplay of the mre11 nuclease and ku in the response to replication-associated DNA damage. Mol Cell Biol 31(21):4379-89 | |
| Refolio E, et al. (2011) The Ddc2/ATRIP checkpoint protein monitors meiotic recombination intermediates. J Cell Sci 124(Pt 14):2488-500 | |
| Reid RJ, et al. (2011) Selective ploidy ablation, a high-throughput plasmid transfer protocol, identifies new genes affecting topoisomerase I-induced DNA damage. Genome Res 21(3):477-86 | |
| Bonetti D, et al. (2010) The MRX complex plays multiple functions in resection of Yku- and Rif2-protected DNA ends. PLoS One 5(11):e14142 | |
| Hamilton NK and Maizels N (2010) MRE11 Function in Response to Topoisomerase Poisons Is Independent of its Function in Double-Strand Break Repair in Saccharomyces cerevisiae. PLoS One 5(10):e15387 | |
| Manfrini N, et al. (2010) Processing of meiotic DNA double strand breaks requires cyclin-dependent kinase and multiple nucleases. J Biol Chem 285(15):11628-37 | |
| Mimitou EP and Symington LS (2010) Ku prevents Exo1 and Sgs1-dependent resection of DNA ends in the absence of a functional MRX complex or Sae2. EMBO J 29(19):3358-69 | |
| Moriel-Carretero M and Aguilera A (2010) A Postincision-Deficient TFIIH Causes Replication Fork Breakage and Uncovers Alternative Rad51- or Pol32-Mediated Restart Mechanisms. Mol Cell 37(5):690-701 | |
| Shim EY, et al. (2010) Saccharomyces cerevisiae Mre11/Rad50/Xrs2 and Ku proteins regulate association of Exo1 and Dna2 with DNA breaks. EMBO J 29(19):3370-80 | |
| Sundararajan R, et al. (2010) Double-strand break repair pathways protect against CAG/CTG repeat expansions, contractions and repeat-mediated chromosomal fragility in Saccharomyces cerevisiae. Genetics 184(1):65-77 | |
| Bonetti D, et al. (2009) Multiple pathways regulate 3' overhang generation at S. cerevisiae telomeres. Mol Cell 35(1):70-81 | |
| Budd ME and Campbell JL (2009) Interplay of Mre11 nuclease with Dna2 plus Sgs1 in Rad51-dependent recombinational repair. PLoS One 4(1):e4267 | |
| Casper AM, et al. (2009) Chromosome aberrations resulting from double-strand DNA breaks at a naturally occurring yeast fragile site composed of inverted ty elements are independent of Mre11p and Sae2p. Genetics 183(2):423-39, 1SI-26SI | |
| Doksani Y, et al. (2009) Replicon dynamics, dormant origin firing, and terminal fork integrity after double-strand break formation. Cell 137(2):247-58 | |
| Enserink JM, et al. (2009) Cdc28/Cdk1 positively and negatively affects genome stability in S. cerevisiae. J Cell Biol 185(3):423-37 | |
| Kim HS, et al. (2008) Functional interactions between Sae2 and the Mre11 complex. Genetics 178(2):711-23 | |
| Lee K, et al. (2008) Saccharomyces cerevisiae ATM orthologue suppresses break-induced chromosome translocations. Nature 454(7203):543-6 | |
| Mimitou EP and Symington LS (2008) Sae2, Exo1 and Sgs1 collaborate in DNA double-strand break processing. Nature 455(7214):770-4 | |
| Santoyo G and Strathern JN (2008) Non-homologous end joining is important for repair of Cr(VI)-induced DNA damage in Saccharomyces cerevisiae. Microbiol Res 163(1):113-9 | |
| Alvaro D, et al. (2007) Genome-wide analysis of Rad52 foci reveals diverse mechanisms impacting recombination. PLoS Genet 3(12):e228 | |
| Curcio MJ, et al. (2007) S-phase checkpoint pathways stimulate the mobility of the retrovirus-like transposon Ty1. Mol Cell Biol 27(24):8874-85 | |
| Lee K and Lee SE (2007) Saccharomyces cerevisiae Sae2- and Tel1-dependent single-strand DNA formation at DNA break promotes microhomology-mediated end joining. Genetics 176(4):2003-14 | |
| Clerici M, et al. (2006) The Saccharomyces cerevisiae Sae2 protein negatively regulates DNA damage checkpoint signalling. EMBO Rep 7(2):212-8 | |
| Wan L, et al. (2006) Chemical inactivation of cdc7 kinase in budding yeast results in a reversible arrest that allows efficient cell synchronization prior to meiotic recombination. Genetics 174(4):1767-74 | |
| Wu HY and Burgess SM (2006) Two distinct surveillance mechanisms monitor meiotic chromosome metabolism in budding yeast. Curr Biol 16(24):2473-9 | |
| Chakhparonian M, et al. (2005) A mutation in yeast Tel1p that causes differential effects on the DNA damage checkpoint and telomere maintenance. Curr Genet 48(5):310-22 |




