Other names published for TAH11: SID2, CDT1, YJR046W
TAH11 LITERATURE TOPICS
- Curated Literature
- Genetics/Cell Biology
- Nucleic Acid Information
- Gene Product Information
- Related Genes/Proteins
- Research Aids
- Genome-wide Analysis
- Other Topics
- Additional Information
TAH11 - Function/Process (12)
| Reference | Other Genes Addressed |
|---|---|
| Wu R, et al. (2012) Cdt1p, through its interaction with Mcm6p, is required for the formation, nuclear accumulation and chromatin loading of the MCM complex. J Cell Sci 125(Pt 1):209-19 | |
| Takara TJ and Bell SP (2011) Multiple Cdt1 molecules act at each origin to load replication-competent Mcm2-7 helicases. EMBO J 30(24):4885-96 | |
| Lydeard JR, et al. (2010) Break-induced replication requires all essential DNA replication factors except those specific for pre-RC assembly. Genes Dev 24(11):1133-44 | |
| Tsakraklides V and Bell SP (2010) Dynamics of pre-replicative complex assembly. J Biol Chem 285(13):9437-43 | |
| Asano T, et al. (2007) Interaction between ORC and Cdt1p of Saccharomyces cerevisiae. FEMS Yeast Res 7(8):1256-62 | |
| Chen S, et al. (2007) Orc6 is required for dynamic recruitment of Cdt1 during repeated Mcm2 7 loading. Genes Dev 21(22):2897-2907 | |
| Kawasaki Y, et al. (2006) Reconstitution of Saccharomyces cerevisiae prereplicative complex assembly in vitro. Genes Cells 11(7):745-56 | |
| Randell JC, et al. (2006) Sequential ATP hydrolysis by Cdc6 and ORC directs loading of the Mcm2-7 helicase. Mol Cell 21(1):29-39 | |
| Devault A, et al. (2002) Identification of Tah11/Sid2 as the ortholog of the replication licensing factor Cdt1 in Saccharomyces cerevisiae. Curr Biol 12(8):689-94 | |
| Maiorano D and Mechali M (2002) Many roads lead to the origin. Nat Cell Biol 4(3):E58-9 | |
| Tanaka S and Diffley JF (2002) Interdependent nuclear accumulation of budding yeast Cdt1 and Mcm2-7 during G1 phase. Nat Cell Biol 4(3):198-207 | |
| Jacobson MD, et al. (2001) Mutations in SID2, a novel gene in Saccharomyces cerevisiae, cause synthetic lethality with sic1 deletion and may cause a defect during S phase. Genetics 159(1):17-33 |




