SGS1/YMR190C Literature Guide Help

Other names published for SGS1: YMR190C

SGS1 - Cross-species Expression (9)

ReferenceOther Genes Addressed
Mirzaei H, et al.  (2011) Sgs1 Truncations Induce Genome Rearrangements but Suppress Detrimental Effects of BLM Overexpression in Saccharomyces cerevisiae. J Mol Biol 405(4):877-91
Chen CF and Brill SJ  (2010) An essential DNA strand-exchange activity is conserved in the divergent N-termini of BLM orthologs. EMBO J 29(10):1713-25
Aggarwal M and Brosh RM  (2009) WRN helicase defective in the premature aging disorder Werner syndrome genetically interacts with topoisomerase 3 and restores the top3 slow growth phenotype of sgs1 top3. Aging (Albany NY) 1(2):219-33
Spillare EA, et al.  (2006) Redundancy of DNA helicases in p53-mediated apoptosis. Oncogene 25(14):2119-23
Lillard-Wetherell K, et al.  (2005) BLM helicase complements disrupted type II telomere lengthening in telomerase-negative sgs1 yeast. Cancer Res 65(13):5520-2
Nakayama M, et al.  (2004) The possible roles of the DNA helicase and C-terminal domains in RECQ5/QE: complementation study in yeast. DNA Repair (Amst) 3(4):369-78
Heo SJ, et al.  (1999) Bloom's syndrome gene suppresses premature ageing caused by Sgs1 deficiency in yeast. Genes Cells 4(11):619-25
Neff NF, et al.  (1999) The DNA helicase activity of BLM is necessary for the correction of the genomic instability of bloom syndrome cells. Mol Biol Cell 10(3):665-76
Yamagata K, et al.  (1998) Bloom's and Werner's syndrome genes suppress hyperrecombination in yeast sgs1 mutant: implication for genomic instability in human diseases. Proc Natl Acad Sci U S A 95(15):8733-8