GLN3/YER040W Literature Guide Help

Other names published for GLN3: YER040W

GLN3 - Cellular Location (45)

ReferenceOther Genes Addressed
Levi CE, et al.  (2012) GABA induction of the Saccharomyces cerevisiae UGA4 gene depends on the quality of the carbon source: role of the key transcription factors acting in this process. Biochem Biophys Res Commun 421(3):572-7
Georis I, et al.  (2011) Intranuclear Function for Protein Phosphatase 2A: Pph21 and Pph22 Are Required for Rapamycin-Induced GATA Factor Binding to the DAL5 Promoter in Yeast. Mol Cell Biol 31(1):92-104
Georis I, et al.  (2011) Nitrogen-responsive regulation of GATA protein family activators Gln3 and Gat1 occurs by two distinct pathways, one inhibited by rapamycin and the other by methionine sulfoximine. J Biol Chem 286(52):44897-912
Hirasaki M, et al.  (2011) Saccharomyces cerevisiae protein phosphatase Ppz1 and protein kinases Sat4 and Hal5 are involved in the control of subcellular localization of Gln3 by likely regulating its phosphorylation state. J Biosci Bioeng 111(3):249-54
Babbitt GA  (2010) Relaxed selection against accidental binding of transcription factors with conserved chromatin contexts. Gene 466(1-2):43-8
Breitkreutz A, et al.  (2010) A global protein kinase and phosphatase interaction network in yeast. Science 328(5981):1043-6
Tate JJ, et al.  (2010) Distinct phosphatase requirements and GATA factor responses to nitrogen catabolite repression and rapamycin treatment in Saccharomyces cerevisiae. J Biol Chem 285(23):17880-95
Georis I, et al.  (2009) Nitrogen Catabolite Repression-Sensitive Transcription as a Readout of Tor Pathway Regulation: The Genetic Background, Reporter Gene and GATA Factor Assayed Determine the Outcomes. Genetics 181(3):861-74
Georis I, et al.  (2009) The yeast GATA factor Gat1 occupies a central position in nitrogen catabolite repression-sensitive gene activation. Mol Cell Biol 29(13):3803-15
Leverentz MK, et al.  (2009) Mutation of a Phosphorylatable Residue in Put3p Affects the Magnitude of Rapamycin-induced PUT1 Activation in a Gat1p-dependent Manner. J Biol Chem 284(36):24115-22
Tate JJ, et al.  (2009) Rapamycin-induced Gln3 Dephosphorylation Is Insufficient for Nuclear Localization: Sit4 AND PP2A PHOSPHATASES ARE REGULATED AND FUNCTION DIFFERENTLY. J Biol Chem 284(4):2522-34
Georis I, et al.  (2008) Tor Pathway Control of the Nitrogen-responsive DAL5 Gene Bifurcates at the Level of Gln3 and Gat1 Regulation in Saccharomyces cerevisiae. J Biol Chem 283(14):8919-29
Hirasaki M, et al.  (2008) Protein phosphatase Siw14 controls intracellular localization of Gln3 in cooperation with Npr1 kinase in Saccharomyces cerevisiae. Gene 409(1-2):34-43
Puria R, et al.  (2008) Nuclear translocation of Gln3 in response to nutrient signals requires Golgi-to-endosome trafficking in Saccharomyces cerevisiae. Proc Natl Acad Sci U S A 105(20):7194-9
Tate JJ and Cooper TG  (2008) Formalin can alter the intracellular localization of some transcription factors in Saccharomyces cerevisiae. FEMS Yeast Res 8(8):1223-35
Luzzani C, et al.  (2007) New insights into the regulation of the Saccharomyces cerevisiae UGA4 gene: two parallel pathways participate in carbon-regulated transcription. Microbiology 153(Pt 11):3677-3684
Tate JJ and Cooper TG  (2007) Stress-responsive Gln3 localization in Saccharomyces cerevisiae is separable from and can overwhelm nitrogen source regulation. J Biol Chem 282(25):18467-80
Cameroni E, et al.  (2006) Phosphatidylinositol 4-Phosphate Is Required for Translation Initiation in Saccharomyces cerevisiae. J Biol Chem 281(50):38139-49
Devasahayam G, et al.  (2006) Pmr1, a Golgi Ca2+/Mn2+-ATPase, is a regulator of the target of rapamycin (TOR) signaling pathway in yeast. Proc Natl Acad Sci U S A 103(47):17840-5
Feller A, et al.  (2006) Transduction of the nitrogen signal activating Gln3-mediated transcription is independent of Npr1 kinase and Rsp5-Bul1/2 ubiquitin ligase in Saccharomyces cerevisiae. J Biol Chem 281(39):28546-54
Kulkarni A, et al.  (2006) Differing responses of Gat1 and Gln3 phosphorylation and localization to rapamycin and methionine sulfoximine treatment in Saccharomyces cerevisiae. FEMS Yeast Res 6(2):218-29
Tate JJ, et al.  (2006) Ammonia-specific regulation of Gln3 localization in Saccharomyces cerevisiae by protein kinase Npr1. J Biol Chem 281(38):28460-9
Tate JJ, et al.  (2006) Saccharomyces cerevisiae Sit4 phosphatase is active irrespective of the nitrogen source provided, and Gln3 phosphorylation levels become nitrogen source-responsive in a sit4-deleted strain. J Biol Chem 281(49):37980-92
Avendano A, et al.  (2005) Swi/SNF-GCN5-dependent chromatin remodelling determines induced expression of GDH3, one of the paralogous genes responsible for ammonium assimilation and glutamate biosynthesis in Saccharomyces cerevisiae. Mol Microbiol 57(1):291-305
Giannattasio S, et al.  (2005) Retrograde response to mitochondrial dysfunction is separable from TOR1/2 regulation of retrograde gene expression. J Biol Chem 280(52):42528-35
Tate JJ, et al.  (2005) Methionine sulfoximine treatment and carbon starvation elicit Snf1-independent phosphorylation of the transcription activator Gln3 in Saccharomyces cerevisiae. J Biol Chem 280(29):27195-204
Cox KH, et al.  (2004) Actin cytoskeleton is required for nuclear accumulation of Gln3 in response to nitrogen limitation but not rapamycin treatment in Saccharomyces cerevisiae. J Biol Chem 279(18):19294-301
Cox KH, et al.  (2004) Gln3 phosphorylation and intracellular localization in nutrient limitation and starvation differ from those generated by rapamycin inhibition of Tor1/2 in Saccharomyces cerevisiae. J Biol Chem 279(11):10270-8
Schmelzle T, et al.  (2004) Activation of the RAS/cyclic AMP pathway suppresses a TOR deficiency in yeast. Mol Cell Biol 24(1):338-51
Carvalho J and Zheng XF  (2003) Domains of Gln3p interacting with karyopherins, Ure2p, and the target of rapamycin protein. J Biol Chem 278(19):16878-86