Other names published for MRC1: YCL060C, YCL061C
MRC1 LITERATURE TOPICS
- Curated Literature
- Additional Literature
- All Curated References
- Primary Literature
- Reviews
- Genetics/Cell Biology
- Nucleic Acid Information
- Gene Product Information
- Related Genes/Proteins
- Research Aids
- Genome-wide Analysis
- Proteome-wide Analysis
- Additional Information
MRC1 - Additional Literature (71)
| Reference | Other Genes Addressed |
|---|---|
| Alver B, et al. (2013) A Whole Genome Screen for Minisatellite Stability Genes in Stationary Phase Yeast Cells. G3 (Bethesda) () | |
| Darvishi E, et al. (2013) Thymol antifungal mode of action involves telomerase inhibition. Med Mycol () | |
| Alzu A, et al. (2012) Senataxin associates with replication forks to protect fork integrity across RNA-polymerase-II-transcribed genes. Cell 151(4):835-46 | |
| Berens TJ and Toczyski DP (2012) Colocalization of Mec1 and Mrc1 is sufficient for Rad53 phosphorylation in vivo. Mol Biol Cell 23(6):1058-67 | |
| Collura A, et al. (2012) Abasic sites linked to dUTP incorporation in DNA are a major cause of spontaneous mutations in absence of base excision repair and Rad17-Mec3-Ddc1 (9-1-1) DNA damage checkpoint clamp in Saccharomyces cerevisiae. DNA Repair (Amst) 11(3):294-303 | |
| De Piccoli G, et al. (2012) Replisome stability at defective DNA replication forks is independent of S phase checkpoint kinases. Mol Cell 45(5):696-704 | |
| Fernandez MA, et al. (2012) Identification of a core set of signature cell cycle genes whose relative order of time to peak expression is conserved across species. Nucleic Acids Res 40(7):2823-32 | |
| Poli J, et al. (2012) dNTP pools determine fork progression and origin usage under replication stress. EMBO J 31(4):883-94 | |
| Wang G, et al. (2012) Multiple phosphorylation of Rad9 by CDK is required for DNA damage checkpoint activation. Cell Cycle 11(20) | |
| Wang G, et al. (2012) Multiple phosphorylation of Rad9 by CDK is required for DNA damage checkpoint activation. Cell Cycle 11(20):3792-800 | |
| Zhang Y, et al. (2012) Genome-wide screen identifies pathways that govern GAA/TTC repeat fragility and expansions in dividing and nondividing yeast cells. Mol Cell 48(2):254-65 | |
| Addinall SG, et al. (2011) Quantitative Fitness Analysis Shows That NMD Proteins and Many Other Protein Complexes Suppress or Enhance Distinct Telomere Cap Defects. PLoS Genet 7(4):e1001362 | |
| Budd ME, et al. (2011) Inviability of a DNA2 deletion mutant is due to the DNA damage checkpoint. Cell Cycle 10(10):1690-8 | |
| Chang HY, et al. (2011) Genome-wide analysis to identify pathways affecting telomere-initiated senescence in budding yeast. G3 (Bethesda) 1(3):197-208 | |
| Chatre L and Ricchetti M (2011) Nuclear Mitochondrial DNA Activates Replication in Saccharomyces cerevisiae. PLoS One 6(3):e17235 | |
| Deshpande AM, et al. (2011) Polymerase epsilon is required to maintain replicative senescence. Mol Cell Biol 31(8):1637-45 | |
| Gellon L, et al. (2011) New Functions of Ctf18-RFC in Preserving Genome Stability outside Its Role in Sister Chromatid Cohesion. PLoS Genet 7(2):e1001298 | |
| Hashash N, et al. (2011) Regulation of fragile sites expression in budding yeast by MEC1, RRM3 and hydroxyurea. J Cell Sci 124(Pt 2):181-5 | |
| Ivanovska I, et al. (2011) Control of chromatin structure by spt6: different consequences in coding and regulatory regions. Mol Cell Biol 31(3):531-41 | |
| Kubota T, et al. (2011) Quantitative proteomic analysis of chromatin reveals that Ctf18 acts in the DNA replication checkpoint. Mol Cell Proteomics 10(7):M110.005561 | |
| Li XC and Tye BK (2011) Ploidy Dictates Repair Pathway Choice under DNA Replication Stress. Genetics 187(4):1031-40 | |
| Minca EC and Kowalski D (2011) Replication fork stalling by bulky DNA damage: localization at active origins and checkpoint modulation. Nucleic Acids Res 39(7):2610-23 | |
| Reha-Krantz LJ, et al. (2011) Drug-sensitive DNA polymerase d reveals a role for mismatch repair in checkpoint activation in yeast. Genetics 189(4):1211-24 | |
| Tripathi K, et al. (2011) Cellular morphogenesis under stress is influenced by the sphingolipid pathway gene ISC1 and DNA integrity checkpoint genes in Saccharomyces cerevisiae. Genetics 189(2):533-47 | |
| Chen SH, et al. (2010) A proteome-wide analysis of kinase-substrate network in the DNA damage response. J Biol Chem 285(17):12803-12 | |
| Crabbe L, et al. (2010) Analysis of replication profiles reveals key role of RFC-Ctf18 in yeast replication stress response. Nat Struct Mol Biol 17(11):1391-1397 | |
| Kile AC and Koepp DM (2010) Activation of the s-phase checkpoint inhibits degradation of the f-box protein dia2. Mol Cell Biol 30(1):160-71 | |
| On T, et al. (2010) The evolutionary landscape of the chromatin modification machinery reveals lineage specific gains, expansions, and losses. Proteins 78(9):2075-89 | |
| Putnam CD, et al. (2010) Post-replication repair suppresses duplication-mediated genome instability. PLoS Genet 6():e1000933 | |
| Chang M, et al. (2009) Telomerase is essential to alleviate pif1-induced replication stress at telomeres. Genetics 183(3):779-91 |




