| 1) |
Neiman A (2007) personal communication
|
| 2) |
Grant CM, et al. (1998) Glutathione and catalase provide overlapping defenses for protection against hydrogen peroxide in the yeast Saccharomyces cerevisiae. Biochem Biophys Res Commun 253(3):893-8
|
| 3) |
Traczyk A, et al. (1985) Catalase T deficient mutants of Saccharomyces cerevisiae. Acta Microbiol Pol 34(3-4):231-41
|
| 4) |
Cohen G, et al. (1985) Isolation of the catalase A gene of Saccharomyces cerevisiae by complementation of the cta1 mutation. Mol Gen Genet 200(1):74-9
|
| 5) |
Petrova VY, et al. (2004) Dual targeting of yeast catalase A to peroxisomes and mitochondria. Biochem J 380(Pt 2):393-400
|
| 6) |
Jamieson DJ (1998) Oxidative stress responses of the yeast Saccharomyces cerevisiae. Yeast 14(16):1511-27
|
| 7) |
Wieser R, et al. (1991) Heat shock factor-independent heat control of transcription of the CTT1 gene encoding the cytosolic catalase T of Saccharomyces cerevisiae. J Biol Chem 266(19):12406-11
|
| 8) |
Lushchak VI and Gospodaryov DV (2005) Catalases protect cellular proteins from oxidative modification in Saccharomyces cerevisiae. Cell Biol Int 29(3):187-92
|
| 9) |
Marchler G, et al. (1993) A Saccharomyces cerevisiae UAS element controlled by protein kinase A activates transcription in response to a variety of stress conditions. EMBO J 12(5):1997-2003
|
| 10) |
Agarwal S, et al. (2005) Caloric restriction augments ROS defense in S. cerevisiae, by a Sir2p independent mechanism. Free Radic Res 39(1):55-62
|
| 11) |
Giannattasio S, et al. (2005) Acid stress adaptation protects Saccharomyces cerevisiae from acetic acid-induced programmed cell death. Gene 354:93-8
|
| 12) |
Davidson JF, et al. (1996) Oxidative stress is involved in heat-induced cell death in Saccharomyces cerevisiae. Proc Natl Acad Sci U S A 93(10):5116-21
|
| 13) |
Herrero E, et al. (2008) Redox control and oxidative stress in yeast cells. Biochim Biophys Acta 1780(11):1217-35
|
| 14) |
Martinez-Pastor MT, et al. (1996) The Saccharomyces cerevisiae zinc finger proteins Msn2p and Msn4p are required for transcriptional induction through the stress response element (STRE). EMBO J 15(9):2227-35
|
| 15) |
Schuller C, et al. (1994) The HOG pathway controls osmotic regulation of transcription via the stress response element (STRE) of the Saccharomyces cerevisiae CTT1 gene. EMBO J 13(18):4382-9
|
| 16) |
Lee J, et al. (1999) Yap1 and Skn7 control two specialized oxidative stress response regulons in yeast. J Biol Chem 274(23):16040-6
|
| 17) |
Wu CY, et al. (2008) Differential control of Zap1-regulated genes in response to zinc deficiency in Saccharomyces cerevisiae. BMC Genomics 9:370
|
| 18) |
Winkler H, et al. (1988) Co-ordinate control of synthesis of mitochondrial and non-mitochondrial hemoproteins: a binding site for the HAP1 (CYP1) protein in the UAS region of the yeast catalase T gene (CTT1). EMBO J 7(6):1799-804
|
| 19) |
Erjavec N and Nystrom T (2007) Sir2p-dependent protein segregation gives rise to a superior reactive oxygen species management in the progeny of Saccharomyces cerevisiae. Proc Natl Acad Sci U S A 104(26):10877-81
|
| 20) |
Cohen G, et al. (1988) Sequence of the Saccharomyces cerevisiae CTA1 gene and amino acid sequence of catalase A derived from it. Eur J Biochem 176(1):159-63
|
| 21) |
Aksam EB, et al. (2009) Preserving organelle vitality: peroxisomal quality control mechanisms in yeast. FEMS Yeast Res 9(6):808-20
|